Compounds > proline

proline and Granulocytic Leukemia, Chronic

proline has been researched along with Granulocytic Leukemia, Chronic in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (50.00)29.6817
2010's2 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Camelo-Santos, J; de Paula Silveira-Lacerda, E; do Prado Barbosa, A; Guillo, LA1
Jaymacker, J; Mowla, S; Njikan, S; Novitzky, N; Shankland, I; Shires, K1
Smith, KM; Van Etten, RA; Yacobi, R1
Dai, Z; Kerzic, P; McNiece, IK; Schroeder, WG1

Other Studies

4 other study(ies) available for proline and Granulocytic Leukemia, Chronic

ArticleYear
Arginine homozygosity in codon 72 of p53 correlates with failure to imatinib response in chronic myeloid leukemia.
    Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 2013, Volume: 67, Issue:2

    Topics: Adult; Alleles; Antineoplastic Agents; Arginine; Benzamides; Codon; Female; Genetic Predisposition to Disease; Genotype; Heterozygote; Homozygote; Humans; Imatinib Mesylate; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Male; Piperazines; Polymorphism, Genetic; Proline; Pyrimidines; Tumor Suppressor Protein p53

2013
Serine and proline-rich ligands enriched via phage-display technology show preferential binding to BCR/ABL expressing cells.
    Hematology/oncology and stem cell therapy, 2014, Volume: 7, Issue:1

    Topics: Amino Acid Sequence; Animals; Cell Surface Display Techniques; Clone Cells; Fibroblasts; Fusion Proteins, bcr-abl; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Ligands; Mice; Molecular Sequence Data; NIH 3T3 Cells; Peptides; Proline; Protein Binding; Sequence Alignment; Serine; Transfection

2014
Autoinhibition of Bcr-Abl through its SH3 domain.
    Molecular cell, 2003, Volume: 12, Issue:1

    Topics: 3T3 Cells; Alanine; Amino Acid Sequence; Animals; Binding Sites; Catalytic Domain; Cell Transformation, Neoplastic; Enzyme Inhibitors; Eukaryotic Cells; Feedback, Physiological; Fusion Proteins, bcr-abl; Humans; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Mice; Mice, Inbred BALB C; Models, Molecular; Mutation; Phosphorylation; Proline; Protein Binding; Protein Structure, Tertiary; Protein-Tyrosine Kinases; Tyrosine

2003
Deletion of the Src homology 3 domain and C-terminal proline-rich sequences in Bcr-Abl prevents Abl interactor 2 degradation and spontaneous cell migration and impairs leukemogenesis.
    The Journal of biological chemistry, 2001, Aug-03, Volume: 276, Issue:31

    Topics: Adaptor Proteins, Signal Transducing; Amino Acid Sequence; Animals; Bone Marrow Transplantation; Cell Line; Chemotaxis; Fibronectins; Fusion Proteins, bcr-abl; Homeodomain Proteins; Humans; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Male; Mice; Mice, Inbred C57BL; Mice, Inbred DBA; Mice, Transgenic; Mutagenesis; Peptide Fragments; Proline; Recombinant Proteins; Retroviridae; Sequence Deletion; src Homology Domains; Transfection; Tumor Cells, Cultured; Ubiquitins

2001