proline has been researched along with Clinically Isolated CNS Demyelinating Syndrome in 11 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 3 (27.27) | 18.7374 |
1990's | 2 (18.18) | 18.2507 |
2000's | 3 (27.27) | 29.6817 |
2010's | 1 (9.09) | 24.3611 |
2020's | 2 (18.18) | 2.80 |
Authors | Studies |
---|---|
Behera, A; Das Sarma, J; Kishore, A; Maity, D; Mulchandani, V; Pal, D; Safiriyu, AA; Singh, M; Sinha, B | 1 |
Das Sarma, J; Rout, SS; Shindler, KS; Singh, M | 1 |
Das Sarma, J; Kenyon, LC; Kishore, A; Krishnarjuna, B; Maity, D; Pal, D; Raghothama, S; Singh, M; Sunanda, P; Vappala, S | 1 |
Bhagavati, S; Maccabee, PJ; Xu, W | 1 |
Khan, M; Pannu, R; Singh, AK; Singh, I; Won, JS | 1 |
Alevra, X; Christodoulou, K; Georgiou, DM; Hadjisavvas, A; Kleopa, KA; Kyriakides, T; Nicolaou, P; Zamba-Papanicolaou, E | 1 |
Ikeda, H; Jenkins, HG | 1 |
Ikeda, H; Jenkins, HG; Kaler, BS | 1 |
Ikeda, H; Tansey, EM | 1 |
Ikeda, H; Jenkins, HG; Macefield, F; Tansey, EM | 1 |
Wajgt, A; Wender, M; Zgorzalewicz, B | 1 |
11 other study(ies) available for proline and Clinically Isolated CNS Demyelinating Syndrome
Article | Year |
---|---|
Two Consecutive Prolines in the Fusion Peptide of Murine β-Coronavirus Spike Protein Predominantly Determine Fusogenicity and May Be Essential but Not Sufficient to Cause Demyelination.
Topics: Animals; Demyelinating Diseases; Mice; Peptides; Proline; Spike Glycoprotein, Coronavirus; Viral Envelope Proteins | 2022 |
One proline deletion in the fusion peptide of neurotropic mouse hepatitis virus (MHV) restricts retrograde axonal transport and neurodegeneration.
Topics: Animals; Axonal Transport; Axons; Brain; Coronavirus Infections; Demyelinating Diseases; Male; Mice; Mice, Inbred C57BL; Murine hepatitis virus; Optic Nerve; Peptides; Proline; Sequence Deletion; Spike Glycoprotein, Coronavirus; Viral Envelope Proteins | 2020 |
A proline insertion-deletion in the spike glycoprotein fusion peptide of mouse hepatitis virus strongly alters neuropathology.
Topics: Animals; Brain; Cell Line; Demyelinating Diseases; INDEL Mutation; Meningitis, Viral; Mice; Murine hepatitis virus; Nuclear Magnetic Resonance, Biomolecular; Proline; Protein Domains; Structure-Activity Relationship; Viral Envelope Proteins | 2019 |
The neurofilament light chain gene (NEFL) mutation Pro22Ser can be associated with mixed axonal and demyelinating neuropathy.
Topics: Adolescent; Amino Acid Sequence; Amino Acid Substitution; Axons; Charcot-Marie-Tooth Disease; Demyelinating Diseases; DNA Mutational Analysis; Electrodiagnosis; Genetic Markers; Genetic Predisposition to Disease; Genotype; Humans; Male; Mutation; Mutation, Missense; Neural Conduction; Neurofilament Proteins; Peripheral Nerves; Phenotype; Proline; Serine | 2009 |
A novel role of lactosylceramide in the regulation of lipopolysaccharide/interferon-gamma-mediated inducible nitric oxide synthase gene expression: implications for neuroinflammatory diseases.
Topics: Acetylcysteine; Animals; Antigens, CD; Antioxidants; Apoptosis; Astrocytes; Cells, Cultured; Demyelinating Diseases; Drug Evaluation, Preclinical; Enzyme Induction; Enzyme Inhibitors; Fatty Acids; Female; Gait Disorders, Neurologic; Galactosyltransferases; I-kappa B Proteins; Inflammation; Interferon-gamma; Lactosylceramides; Lipopolysaccharides; Morpholines; Nerve Tissue Proteins; NF-kappa B; Nitric Oxide; Nitric Oxide Synthase; Nitric Oxide Synthase Type II; Oxidation-Reduction; Phosphorylation; Proline; Protein Processing, Post-Translational; Rats; Rats, Sprague-Dawley; Recombinant Proteins; Signal Transduction; Spinal Cord Injuries; Thiocarbamates; Transfection | 2004 |
Phenotypic and cellular expression of two novel connexin32 mutations causing CMT1X.
Topics: Aged; Amino Acid Substitution; Central Nervous System Diseases; Charcot-Marie-Tooth Disease; Connexins; Demyelinating Diseases; Electromyography; Female; Gap Junction beta-1 Protein; Glutamic Acid; HeLa Cells; Humans; Leucine; Male; Middle Aged; Mutation, Missense; Neural Conduction; Pedigree; Phenotype; Polyneuropathies; Proline; Sural Nerve; Valine | 2006 |
Tumour necrosis factor causes an increase in axonal transport of protein and demyelination in the mouse optic nerve.
Topics: Animals; Axonal Transport; Demyelinating Diseases; Injections; Male; Mice; Nerve Tissue Proteins; Optic Nerve; Proline; Recombinant Proteins; Stimulation, Chemical; Superior Colliculi; Tumor Necrosis Factor-alpha; Vitreous Body | 1992 |
Changes in fast axonal transport of proteins prior to demyelination in optic nerves of mice following Semliki Forest virus infection.
Topics: Animals; Axonal Transport; Demyelinating Diseases; Mice; Mice, Inbred Strains; Molecular Weight; Nerve Tissue Proteins; Optic Nerve; Proline; Semliki forest virus; Togaviridae Infections | 1991 |
The relationship between axonal transport of protein and demyelination in the optic nerves of mice infected with Semliki Forest virus.
Topics: Animals; Axonal Transport; Demyelinating Diseases; Male; Mice; Optic Nerve; Proline; Proteins; Retina; Semliki forest virus; Togaviridae Infections | 1986 |
Evidence for a T-cell related factor as the cause of demyelination in mice following Semliki Forest virus infection.
Topics: Animals; Axonal Transport; Demyelinating Diseases; Male; Mice; Mice, Nude; Optic Nerve; Proline; Semliki forest virus; T-Lymphocytes; Togaviridae Infections | 1988 |
Protein synthesis in vitro by the rat brain in course of experimental cyanide encephalopathy.
Topics: Animals; Brain; Brain Diseases; Cyanides; Demyelinating Diseases; Disease Models, Animal; Female; Hypoxia; Leucine; Male; Myelin Sheath; Nerve Tissue Proteins; Poisoning; Proline; Rats | 1974 |