prednisone has been researched along with Thyrotoxicosis in 17 studies
Prednisone: A synthetic anti-inflammatory glucocorticoid derived from CORTISONE. It is biologically inert and converted to PREDNISOLONE in the liver.
prednisone : A synthetic glucocorticoid drug that is particularly effective as an immunosuppressant, and affects virtually all of the immune system. Prednisone is a prodrug that is converted by the liver into prednisolone (a beta-hydroxy group instead of the oxo group at position 11), which is the active drug and also a steroid.
Thyrotoxicosis: A hypermetabolic syndrome caused by excess THYROID HORMONES which may come from endogenous or exogenous sources. The endogenous source of hormone may be thyroid HYPERPLASIA; THYROID NEOPLASMS; or hormone-producing extrathyroidal tissue. Thyrotoxicosis is characterized by NERVOUSNESS; TACHYCARDIA; FATIGUE; WEIGHT LOSS; heat intolerance; and excessive SWEATING.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Type 2 amiodarone-induced thyrotoxicosis (AIT) is a destructive thyroiditis usually responsive to glucocorticoids." | 9.15 | Continuation of amiodarone delays restoration of euthyroidism in patients with type 2 amiodarone-induced thyrotoxicosis treated with prednisone: a pilot study. ( Bartalena, L; Bogazzi, F; Brogioni, S; Martino, E; Rossi, G; Tomisti, L, 2011) |
| "Amiodarone-induced thyrotoxicosis (AIT) may occur either in the presence of underlying thyroid disease (type I AIT) or in apparently normal thyroid glands (type II AIT)." | 9.10 | Treatment of type II amiodarone-induced thyrotoxicosis by either iopanoic acid or glucocorticoids: a prospective, randomized study. ( Aghini-Lombardi, F; Bartalena, L; Bogazzi, F; Braverman, LE; Brogioni, S; Cosci, C; Dell'Unto, E; Grasso, L; Martino, E; Pinchera, A; Rossi, G, 2003) |
| "Type 2 amiodarone-induced thyrotoxicosis (AIT) is a destructive thyroiditis usually responsive to glucocorticoids; however, recent surveys showed that many expert thyroidologists worldwide use thionamides for type 2 AIT patients." | 7.75 | Glucocorticoids are preferable to thionamides as first-line treatment for amiodarone-induced thyrotoxicosis due to destructive thyroiditis: a matched retrospective cohort study. ( Bartalena, L; Bogazzi, F; Dell'Unto, E; Martino, E; Pepe, P; Rossi, G; Tomisti, L, 2009) |
| "This study sought to determine the clinical course and predictors of long-term outcome in patients with documented amiodarone-induced thyrotoxicosis (AIT)." | 7.74 | Amiodarone-induced thyrotoxicosis: clinical course and predictors of outcome. ( Ammann, P; Bilz, S; Conen, D; Kaufmann, C; Melly, L; Muller, B; Osswald, S; Schaer, B; Sticherling, C, 2007) |
| "Amiodarone-induced thyrotoxicosis (AIT) occurs in both abnormal (type I) and apparently normal (type II) thyroid glands due to iodine-induced excessive thyroid hormone synthesis in patients with nodular goiter or latent Graves' disease (type I) or to a thyroid-destructive process caused by amiodarone or iodine (type II)." | 7.69 | Treatment of amiodarone-induced thyrotoxicosis, a difficult challenge: results of a prospective study. ( Bartalena, L; Bogazzi, F; Brogioni, S; Burelli, A; Grasso, L; Martino, E, 1996) |
| "Treatment of amiodarone iodine-induced thyrotoxicosis is often unsuccessful." | 7.67 | Rapid effectiveness of prednisone and thionamides combined therapy in severe amiodarone iodine-induced thyrotoxicosis. Comparison of two groups of patients with apparently normal thyroid glands. ( Barbier, Y; Bornet, H; Broussolle, C; Ducottet, X; Martin, C; Noel, G; Orgiazzi, J, 1989) |
| "Amiodarone-induced thyrotoxicosis (AIT) may occur either in the presence of underlying thyroid disease (type I AIT) or in apparently normal thyroid glands (type II AIT)." | 5.10 | Treatment of type II amiodarone-induced thyrotoxicosis by either iopanoic acid or glucocorticoids: a prospective, randomized study. ( Aghini-Lombardi, F; Bartalena, L; Bogazzi, F; Braverman, LE; Brogioni, S; Cosci, C; Dell'Unto, E; Grasso, L; Martino, E; Pinchera, A; Rossi, G, 2003) |
| "Type 2 amiodarone-induced thyrotoxicosis (AIT) is a destructive thyroiditis usually responsive to glucocorticoids; however, recent surveys showed that many expert thyroidologists worldwide use thionamides for type 2 AIT patients." | 3.75 | Glucocorticoids are preferable to thionamides as first-line treatment for amiodarone-induced thyrotoxicosis due to destructive thyroiditis: a matched retrospective cohort study. ( Bartalena, L; Bogazzi, F; Dell'Unto, E; Martino, E; Pepe, P; Rossi, G; Tomisti, L, 2009) |
| "This study sought to determine the clinical course and predictors of long-term outcome in patients with documented amiodarone-induced thyrotoxicosis (AIT)." | 3.74 | Amiodarone-induced thyrotoxicosis: clinical course and predictors of outcome. ( Ammann, P; Bilz, S; Conen, D; Kaufmann, C; Melly, L; Muller, B; Osswald, S; Schaer, B; Sticherling, C, 2007) |
| "Amiodarone-induced thyrotoxicosis (AIT) occurs in both abnormal (type I) and apparently normal (type II) thyroid glands due to iodine-induced excessive thyroid hormone synthesis in patients with nodular goiter or latent Graves' disease (type I) or to a thyroid-destructive process caused by amiodarone or iodine (type II)." | 3.69 | Treatment of amiodarone-induced thyrotoxicosis, a difficult challenge: results of a prospective study. ( Bartalena, L; Bogazzi, F; Brogioni, S; Burelli, A; Grasso, L; Martino, E, 1996) |
| "Treatment of amiodarone iodine-induced thyrotoxicosis is often unsuccessful." | 3.67 | Rapid effectiveness of prednisone and thionamides combined therapy in severe amiodarone iodine-induced thyrotoxicosis. Comparison of two groups of patients with apparently normal thyroid glands. ( Barbier, Y; Bornet, H; Broussolle, C; Ducottet, X; Martin, C; Noel, G; Orgiazzi, J, 1989) |
| "We report a case of autoimmune thrombocytopenic purpura associated with hyperthyroidism in which the patient's thrombocytopenia and thyrotoxicosis resolved concomitantly." | 2.40 | Autoimmune thrombocytopenic purpura associated with hyperthyroidism in a single individual. ( Aggarwal, A; Doolittle, G, 1997) |
| "Thymic hyperplasia is associated with Graves' disease, particularly in young patients." | 1.36 | Graves' disease and thymic hyperplasia: the relationship of thymic volume to thyroid function. ( Burman, KD; Carroll, NM; Devdhar, M; Popoveniuc, G; Sharma, M; Wartofsky, L; Wexler, JA, 2010) |
| "Pericardial effusion is considered a complication of hypothyroidism; as an expression of thyrotoxicosis, it is extremely rare." | 1.34 | Pericardial effusion as an expression of thyrotoxicosis. ( Lysyy, L; Ovadia, S; Zubkov, T, 2007) |
| "Prednisone was added after another episode of paralysis." | 1.30 | Normokalemic thyrotoxic periodic paralysis: a new therapeutic strategy. ( González-Treviño, O; Rosas-Guzmán, J, 1999) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (11.76) | 18.7374 |
| 1990's | 3 (17.65) | 18.2507 |
| 2000's | 5 (29.41) | 29.6817 |
| 2010's | 7 (41.18) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Ma, C | 1 |
| Hodi, FS | 1 |
| Giobbie-Hurder, A | 1 |
| Wang, X | 1 |
| Zhou, J | 1 |
| Zhang, A | 1 |
| Zhou, Y | 1 |
| Mao, F | 1 |
| Angell, TE | 1 |
| Andrews, CP | 1 |
| Hu, J | 1 |
| Barroso-Sousa, R | 1 |
| Kaiser, UB | 1 |
| Tolaney, SM | 1 |
| Min, L | 1 |
| Bogazzi, F | 4 |
| Tomisti, L | 2 |
| Rossi, G | 3 |
| Dell'Unto, E | 2 |
| Pepe, P | 1 |
| Bartalena, L | 4 |
| Martino, E | 4 |
| Piñar, D | 1 |
| Boix, E | 1 |
| Meana, JA | 1 |
| Herrero, J | 1 |
| Pepene, CE | 1 |
| Seck, T | 1 |
| Diel, I | 1 |
| Minne, HW | 1 |
| Ziegler, R | 1 |
| Pfeilschifter, J | 1 |
| Barbaro, D | 1 |
| Popoveniuc, G | 1 |
| Sharma, M | 1 |
| Devdhar, M | 1 |
| Wexler, JA | 1 |
| Carroll, NM | 1 |
| Wartofsky, L | 1 |
| Burman, KD | 1 |
| Barzilay-Yoseph, L | 1 |
| Shabun, A | 1 |
| Shilo, L | 1 |
| Hadary, R | 1 |
| Nabriski, D | 1 |
| Kitay-Cohen, Y | 1 |
| Brogioni, S | 3 |
| Masannat, Y | 1 |
| Gandhy, R | 1 |
| Olajide, O | 1 |
| Kheetan, R | 1 |
| Yaqub, A | 1 |
| Cosci, C | 1 |
| Grasso, L | 2 |
| Aghini-Lombardi, F | 1 |
| Pinchera, A | 1 |
| Braverman, LE | 1 |
| Sataline, LR | 1 |
| Powell, C | 1 |
| Hamwi, GJ | 1 |
| Ovadia, S | 1 |
| Lysyy, L | 1 |
| Zubkov, T | 1 |
| Conen, D | 1 |
| Melly, L | 1 |
| Kaufmann, C | 1 |
| Bilz, S | 1 |
| Ammann, P | 1 |
| Schaer, B | 1 |
| Sticherling, C | 1 |
| Muller, B | 1 |
| Osswald, S | 1 |
| Burelli, A | 1 |
| Aggarwal, A | 1 |
| Doolittle, G | 1 |
| González-Treviño, O | 1 |
| Rosas-Guzmán, J | 1 |
| Broussolle, C | 1 |
| Ducottet, X | 1 |
| Martin, C | 1 |
| Barbier, Y | 1 |
| Bornet, H | 1 |
| Noel, G | 1 |
| Orgiazzi, J | 1 |
1 review available for prednisone and Thyrotoxicosis
| Article | Year |
|---|---|
Autoimmune thrombocytopenic purpura associated with hyperthyroidism in a single individual.
Topics: Adrenergic beta-Antagonists; Female; gamma-Globulins; Glucocorticoids; Humans; Hyperthyroidism; Inje | 1997 |
2 trials available for prednisone and Thyrotoxicosis
| Article | Year |
|---|---|
Continuation of amiodarone delays restoration of euthyroidism in patients with type 2 amiodarone-induced thyrotoxicosis treated with prednisone: a pilot study.
Topics: Aged; Aged, 80 and over; Amiodarone; Anti-Arrhythmia Agents; Cohort Studies; Female; Glucocorticoids | 2011 |
Treatment of type II amiodarone-induced thyrotoxicosis by either iopanoic acid or glucocorticoids: a prospective, randomized study.
Topics: Aged; Amiodarone; Anti-Arrhythmia Agents; Enzyme Inhibitors; Female; Glucocorticoids; Humans; Iodide | 2003 |
14 other studies available for prednisone and Thyrotoxicosis
| Article | Year |
|---|---|
The Impact of High-Dose Glucocorticoids on the Outcome of Immune-Checkpoint Inhibitor-Related Thyroid Disorders.
Topics: Adult; Aged; Aged, 80 and over; Anti-Inflammatory Agents; Antineoplastic Agents, Immunological; Fema | 2019 |
Glucocorticoids are preferable to thionamides as first-line treatment for amiodarone-induced thyrotoxicosis due to destructive thyroiditis: a matched retrospective cohort study.
Topics: Academic Medical Centers; Adult; Aged; Aged, 80 and over; Amiodarone; Anti-Arrhythmia Agents; Antith | 2009 |
Sunitinib-induced thyrotoxicosis.
Topics: Aged; Antineoplastic Agents; Carbimazole; Carcinoma, Renal Cell; Humans; Indoles; Kidney Neoplasms; | 2009 |
Effect of glucocorticoid-, parathyroid- and thyroid hormones excess on human iliac crest bone matrix insulin-like growth factor (IGF)-I in patients with osteoporosis.
Topics: Bone Density; Bone Matrix; Female; Glucocorticoids; Humans; Hyperparathyroidism; Ilium; Insulin-Like | 2010 |
Sorafenib and thyrotoxicosis.
Topics: Benzenesulfonates; Humans; Liver Neoplasms; Male; Middle Aged; Niacinamide; Phenylurea Compounds; Pr | 2010 |
Graves' disease and thymic hyperplasia: the relationship of thymic volume to thyroid function.
Topics: Adrenergic beta-Antagonists; Adult; Anti-Inflammatory Agents; Antithyroid Agents; Calcium Channel Bl | 2010 |
Thyrotoxic hepatitis.
Topics: Adult; Antithyroid Agents; Biopsy; Diagnosis, Differential; Dose-Response Relationship, Drug; Drug T | 2011 |
Chorea associated with thyrotoxicosis due to toxic multinodular goiter.
Topics: Antithyroid Agents; Atenolol; Chorea; Dibenzothiazepines; Female; Goiter, Nodular; Humans; Hyperthyr | 2011 |
Suppression of the hypercalcemia of thyrotoxicosis by corticosteroids.
Topics: Adrenal Cortex Hormones; Calcium; Cardiovascular Agents; Cortisone; Glucocorticoids; Humans; Hyperca | 1962 |
Pericardial effusion as an expression of thyrotoxicosis.
Topics: Adrenergic beta-Antagonists; Aged; Anti-Inflammatory Agents; Antithyroid Agents; Diuretics; Female; | 2007 |
Amiodarone-induced thyrotoxicosis: clinical course and predictors of outcome.
Topics: Aged; Amiodarone; Anti-Arrhythmia Agents; Diagnosis, Differential; Female; Glucocorticoids; Humans; | 2007 |
Treatment of amiodarone-induced thyrotoxicosis, a difficult challenge: results of a prospective study.
Topics: Adult; Aged; Amiodarone; Drug Therapy, Combination; Female; Humans; Interleukin-6; Male; Methimazole | 1996 |
Normokalemic thyrotoxic periodic paralysis: a new therapeutic strategy.
Topics: Adult; Drug Therapy, Combination; Humans; Male; Methimazole; Paralysis; Potassium; Prednisone; Propr | 1999 |
Rapid effectiveness of prednisone and thionamides combined therapy in severe amiodarone iodine-induced thyrotoxicosis. Comparison of two groups of patients with apparently normal thyroid glands.
Topics: Adult; Aged; Amiodarone; Carbimazole; Drug Therapy, Combination; Female; Humans; Male; Middle Aged; | 1989 |