Compounds > plicamycin
Page last updated: 2024-08-23

plicamycin and Colorectal Cancer

plicamycin has been researched along with Colorectal Cancer in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (60.00)24.3611
2020's2 (40.00)2.80

Authors

AuthorsStudies
Dutta, R; Green, R; Katiri, S; Mohapatra, S; Mohapatra, SS; Patel, B; Quarni, W1
Aoki, M; Hosoda, W; Komori, K; Sakuma, K; Sasaki, E; Shimizu, Y; Yatabe, Y1
Bharadwaj, S; Dutta, R; Green, R; Howell, M; Khalil, R; Mayilsamy, K; Mohapatra, S; Mohapatra, SS1
Bai, Y; Chen, Y; Cheng, T; Gong, W; Guo, Z; Jiang, B; Ma, F; Wang, J; Wu, Y; Zhang, W; Zhang, Y; Zhao, Y; Zhi, F1
Ding, WF; Li, HQ; Shao, KK; Wang, GH; Wang, HM; Wang, XD; Xu, J1

Other Studies

5 other study(ies) available for plicamycin and Colorectal Cancer

ArticleYear
Mithramycin A Inhibits Colorectal Cancer Growth by Targeting Cancer Stem Cells.
    Scientific reports, 2019, 10-23, Volume: 9, Issue:1

    Topics: Animals; Antineoplastic Agents; Cell Line, Tumor; Cell Proliferation; Colorectal Neoplasms; HCT116 Cells; HT29 Cells; Humans; Mice, Inbred C57BL; Neoplastic Stem Cells; Plicamycin

2019
MYB mediates downregulation of the colorectal cancer metastasis suppressor heterogeneous nuclear ribonucleoprotein L-like during epithelial-mesenchymal transition.
    Cancer science, 2021, Volume: 112, Issue:9

    Topics: Binding Sites; Cell Proliferation; Colorectal Neoplasms; Disease Progression; Down-Regulation; Epithelial-Mesenchymal Transition; Gene Expression Regulation, Neoplastic; Gene Knockdown Techniques; Gene Knockout Techniques; Heterogeneous-Nuclear Ribonucleoproteins; HT29 Cells; Humans; Neoplasm Metastasis; Plicamycin; Promoter Regions, Genetic; Proto-Oncogene Proteins c-myb; Sp1 Transcription Factor; Transcription, Genetic; Transfection

2021
Combination Therapy of Mithramycin A and Immune Checkpoint Inhibitor for the Treatment of Colorectal Cancer in an Orthotopic Murine Model.
    Frontiers in immunology, 2021, Volume: 12

    Topics: Animals; Antineoplastic Combined Chemotherapy Protocols; Colorectal Neoplasms; Disease Models, Animal; Female; Immune Checkpoint Inhibitors; Mice; Mice, Inbred C57BL; Plicamycin

2021
Inhibition of the transcription factor Sp1 suppresses colon cancer stem cell growth and induces apoptosis in vitro and in nude mouse xenografts.
    Oncology reports, 2013, Volume: 30, Issue:4

    Topics: AC133 Antigen; Activated-Leukocyte Cell Adhesion Molecule; Animals; Antigens, CD; Apoptosis; Biomarkers, Tumor; Cell Line, Tumor; Cell Proliferation; Colorectal Neoplasms; Down-Regulation; Glycoproteins; HCT116 Cells; Heterografts; HT29 Cells; Humans; Hyaluronan Receptors; Mice; Mice, Inbred BALB C; Mice, Nude; Neoplasm Transplantation; Neoplastic Stem Cells; Peptides; Plicamycin; RNA Interference; RNA, Small Interfering; Sp1 Transcription Factor; Spheroids, Cellular; Up-Regulation

2013
Transcription of promoter from the human APRIL gene regulated by Sp1 and NF-kB.
    Neoplasma, 2012, Volume: 59, Issue:3

    Topics: Base Sequence; Binding Sites; Colorectal Neoplasms; Electrophoretic Mobility Shift Assay; Gene Expression Regulation, Neoplastic; Humans; Luciferases; Molecular Sequence Data; NF-kappa B; Nitriles; Plicamycin; Promoter Regions, Genetic; Protein Binding; Response Elements; Sequence Deletion; Sp1 Transcription Factor; Sulfones; Transcription, Genetic; Tumor Cells, Cultured; Tumor Necrosis Factor Ligand Superfamily Member 13

2012