Page last updated: 2024-08-23

plicamycin and Adenocarcinoma, Basal Cell

plicamycin has been researched along with Adenocarcinoma, Basal Cell in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's3 (75.00)29.6817
2010's1 (25.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chang, WC; Chen, SY; Hsu, TI; Huang, ST; Hung, JJ; Su, WC; Wang, MC; Yeh, YM1
Hisatsune, A; Isohama, Y; Kim, KC; Kuwahara, I; Lillehoj, EP; Lu, W; Miyata, T1
Jia, Z; Le, X; Li, Q; Wang, L; Wei, D; Xie, K; Yao, J; Yuan, P; Zhang, J1
Jia, Z; Le, X; Li, Q; Wang, H; Wang, L; Wei, D; Xie, K; Yao, J; Yuan, P; Zhang, J1

Other Studies

4 other study(ies) available for plicamycin and Adenocarcinoma, Basal Cell

ArticleYear
Betulinic acid decreases specificity protein 1 (Sp1) level via increasing the sumoylation of sp1 to inhibit lung cancer growth.
    Molecular pharmacology, 2012, Volume: 82, Issue:6

    Topics: Adenocarcinoma; Adenocarcinoma of Lung; Animals; Betulinic Acid; Cell Cycle; Cell Cycle Checkpoints; Cell Line, Tumor; Cell Proliferation; Cyclin A2; Cysteine Endopeptidases; Down-Regulation; Endopeptidases; Female; HeLa Cells; Humans; Lung Neoplasms; Mice; Mice, Inbred BALB C; Mice, Nude; Mice, Transgenic; Nuclear Proteins; Pentacyclic Triterpenes; Phosphorylation; Plicamycin; Proteasome Endopeptidase Complex; Retinoblastoma Protein; Sp1 Transcription Factor; Sumoylation; Transcription Factors; Transcriptome; Triterpenes; Ubiquitin; Ubiquitin-Protein Ligases; Xenograft Model Antitumor Assays

2012
Neutrophil elastase stimulates MUC1 gene expression through increased Sp1 binding to the MUC1 promoter.
    American journal of physiology. Lung cellular and molecular physiology, 2005, Volume: 289, Issue:2

    Topics: Adenocarcinoma; Binding Sites; Dactinomycin; Electrophoretic Mobility Shift Assay; Gene Expression Regulation, Neoplastic; Humans; Leukocyte Elastase; Luciferases; Lung Neoplasms; Mucin-1; Plicamycin; Promoter Regions, Genetic; Protein Synthesis Inhibitors; Response Elements; RNA Stability; Sp1 Transcription Factor; Transcription Initiation Site; Transcription, Genetic; Transfection; Tumor Cells, Cultured

2005
Molecular basis of the synergistic antiangiogenic activity of bevacizumab and mithramycin A.
    Cancer research, 2007, May-15, Volume: 67, Issue:10

    Topics: Adenocarcinoma; Angiogenesis Inhibitors; Animals; Antibodies, Monoclonal; Antibodies, Monoclonal, Humanized; Bevacizumab; Cell Line, Tumor; Drug Synergism; Female; Gene Expression; Humans; Mice; Mice, Inbred BALB C; Mice, Nude; Neovascularization, Pathologic; Pancreatic Neoplasms; Plicamycin; Promoter Regions, Genetic; Sp1 Transcription Factor; Up-Regulation; Vascular Endothelial Growth Factor A; Xenograft Model Antitumor Assays

2007
Therapeutic inhibition of Sp1 expression in growing tumors by mithramycin a correlates directly with potent antiangiogenic effects on human pancreatic cancer.
    Cancer, 2007, Dec-15, Volume: 110, Issue:12

    Topics: Adenocarcinoma; Angiogenesis Inhibitors; Animals; Cell Line, Tumor; Female; Humans; Mice; Mice, Inbred C57BL; Mice, Nude; Pancreatic Neoplasms; Plicamycin; RNA, Small Interfering; Sp1 Transcription Factor; Xenograft Model Antitumor Assays

2007