Page last updated: 2024-08-24

plerixafor and Minimal Disease, Residual

plerixafor has been researched along with Minimal Disease, Residual in 2 studies

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Fujita, K; Hatano, K; Kaneda, Y; Murakami, K; Nagahara, A; Nakai, Y; Nakayama, M; Nimura, K; Nonomura, N; Tsuchiya, M; Uemura, M; Yamaguchi, S1
DiPersio, JF; Fruehauf, S; Hanrahan, J; Ho, AD; Hsu, FJ1

Reviews

1 review(s) available for plerixafor and Minimal Disease, Residual

ArticleYear
Relevance and clinical implications of tumor cell mobilization in the autologous transplant setting.
    Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation, 2011, Volume: 17, Issue:7

    Topics: Antineoplastic Combined Chemotherapy Protocols; Benzylamines; Biomarkers, Tumor; Combined Modality Therapy; Cyclams; Cytokines; DNA, Neoplasm; Flow Cytometry; Granulocyte Colony-Stimulating Factor; Hematologic Neoplasms; Hematopoietic Stem Cell Mobilization; Heterocyclic Compounds; Humans; Neoplasm, Residual; Neoplastic Cells, Circulating; Neoplastic Stem Cells; Peripheral Blood Stem Cell Transplantation; Polymerase Chain Reaction; Recurrence; Risk; Translocation, Genetic; Transplantation, Autologous; Treatment Outcome

2011

Other Studies

1 other study(ies) available for plerixafor and Minimal Disease, Residual

ArticleYear
Residual prostate cancer cells after docetaxel therapy increase the tumorigenic potential via constitutive signaling of CXCR4, ERK1/2 and c-Myc.
    Molecular cancer research : MCR, 2013, Volume: 11, Issue:9

    Topics: Animals; Antineoplastic Agents; Benzylamines; Carcinogenesis; Cell Line, Tumor; Cyclams; Disease Models, Animal; Docetaxel; Drug Resistance, Neoplasm; Gene Expression Regulation, Neoplastic; Heterocyclic Compounds; Humans; Male; MAP Kinase Signaling System; Mice, Nude; Mice, SCID; Neoplasm, Residual; Prostatic Neoplasms; Proto-Oncogene Proteins c-myc; Receptors, CXCR4; Signal Transduction; Taxoids

2013