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pk 11195 and Brain Infarction

pk 11195 has been researched along with Brain Infarction in 2 studies

PK-11195 : A monocarboxylic acid amide obtained by formal condensation of the carboxy group of 1-(2-chlorophenyl)isoquinoline-3-carboxylic acid with the amino group of sec-butylmethylamine

Brain Infarction: Tissue NECROSIS in any area of the brain, including the CEREBRAL HEMISPHERES, the CEREBELLUM, and the BRAIN STEM. Brain infarction is the result of a cascade of events initiated by inadequate blood flow through the brain that is followed by HYPOXIA and HYPOGLYCEMIA in brain tissue. Damage may be temporary, permanent, selective or pan-necrosis.

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (50.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Schroeter, M2
Dennin, MA1
Walberer, M2
Backes, H2
Neumaier, B2
Fink, GR2
Graf, R2
Rueger, MA1
Muesken, S1
Jantzen, SU1
Schnakenburg, K1
Hoehn, M1

Other Studies

2 other studies available for pk 11195 and Brain Infarction

ArticleYear
Neuroinflammation extends brain tissue at risk to vital peri-infarct tissue: a double tracer [11C]PK11195- and [18F]FDG-PET study.
    Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism, 2009, Volume: 29, Issue:6

    Topics: Animals; Brain Infarction; Carbon Radioisotopes; Disease Models, Animal; Fluorodeoxyglucose F18; Iso

2009
Effects of minocycline on endogenous neural stem cells after experimental stroke.
    Neuroscience, 2012, Jul-26, Volume: 215

    Topics: 2',3'-Cyclic-Nucleotide Phosphodiesterases; Animals; Anti-Bacterial Agents; Brain Infarction; Brain

2012