pituitrin and Urinary-Retention
pituitrin has been researched along with Urinary-Retention* in 3 studies
Other Studies
3 other study(ies) available for pituitrin and Urinary-Retention
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Dilatative uropathy as a manifestation of neurohypophyseal diabetes insipidus due to a novel mutation in the arginine vasopressin-neurophysin-II gene.
Polydypsia and polyuria are frequent symptoms in patients with sellar masses caused by neurohypophyseal diabetes insipidus. Autosomal dominant familial neurohypophyseal diabetes insipidus (adFNDI), a disorder caused by mutations in the arginine vasopressin (AVP) -neurophysin II (NPII) gene, should be considered as a rare differential diagnosis. A delayed diagnosis bears the risk of life-threatening electrolyte imbalances and permanent urinary tract damage, leading to impaired quality of life.We present a Caucasian kindred of at least 4 generations with FNDI.Clinical histories, endocrine para-meters, and results of molecular analyses of the AVP gene are presented with a review of the literature on diabetes insipidus (DI) related urinary tract dilatation.Polyuria and polydipsia were only reported based on explicit and thorough interrogation after more than 4 years of clinical follow-up. A novel heterozygous mutation in the AVP gene was found in all examined symptomatic subjects (c.1-33_c.4del37nt). A literature review revealed that non-obstructive hydronephrosis (NOH) is a rare but known complication of DI.Since increased fluid intake is often a typical familial pattern in adFNDI, it is frequently missed as being pathologic in affected patients, therefore a detailed clinical history of drinking volumes is of critical importance. AVP gene testing is an important component in the confirmation of the diagnosis. Otherwise unexplainable NOH should lead to further investigations and evaluation of rare diseases like FNDI. Topics: Arginine Vasopressin; Child; Delayed Diagnosis; Diabetes Insipidus; Dilatation, Pathologic; DNA Mutational Analysis; Follow-Up Studies; Humans; Hydronephrosis; Kidney Function Tests; Kidney Pelvis; Magnetic Resonance Imaging; Male; Neurophysins; Pedigree; Protein Precursors; Sequence Analysis, DNA; Ureter; Urinary Bladder; Urinary Retention; Vasopressins | 2013 |
Circadian antidiuretic hormone variation in elderly men complaining of persistent nocturia after urinary flow obstruction removal.
Persistence of nocturia after prostatic resection in healthy patients without symptoms referred to residual bladder instability and to pathological polyuria seems to be caused by an increased urine production at night. The more accreditate mechanism involved is the relevant decreased ADH secretion pattern which occurs at night. In our study, patients with nocturnal poliuria showed significantly low plasmatic vasopressin levels compared with a control group. The aim of this study was to evaluate whether the persistence of nocturia after prostatic resection in healthy patients, without symptoms referred due to residual bladder instability and important polyuria, could be due to a decrease or a lack of increase in antidiuretic hormone (ADH) nocturnal levels following increased urine production at night. Serum ADH, atrial natriuretic peptide (ANP) and osmolality were assessed at 4-h intervals in 12 patients complaining of residual nocturia (group A) and in a control group of 13 patients who had undergone a complete resolution of nocturia after prostate ablation (group B). In the 25 patients involved in the study (mean age 65.8 years), no significant differences were observed in the two groups concerning mean age (67.5 years for group A, 64 years for group B). Mean nocturnal urine volume (1080 +/- 490 ml) in group A patients was significantly higher than in group B (500 +/- 100 ml) (p < 0.001), while no significant differences were found in diurnal diuresis. Mean plasma vasopressin levels of the 12 patients showing an increased nocturnal micturition were found to be significantly lower at all 4-h intervals when compared with the control group (p < 0.05). Individual fluctuations in serum osmolality were slight and insignificant within the normal range in all patients. The diurnal variation of plasma atrial natriuretic peptide was within the reference limits for all subjects during the 24-h period. Our results lead us to believe that residual nocturia after prostatic resection seems to be caused by an increased urine production at night due to a decreased ADH secretion pattern. Topics: Aged; Atrial Natriuretic Factor; Case-Control Studies; Circadian Rhythm; Diuresis; Humans; Male; Osmolar Concentration; Prostatectomy; Urinary Retention; Urination Disorders; Vasopressins | 1998 |
[The effects of renin, angiotensin II, antidiuretic hormone (ADH) and alpha-atrial natriuretic peptide (alpha-ANP) on renal dysfunction induced by IPPV with PEEP].
We investigated the effects of hormonal changes on decreased urine output induced by IPPV with PEEP in 8 dogs anesthetized with nitrous oxide-oxygen-pentobarbital. The decreased urine output could be attributed to the hemodynamic changes as previously reported. The results of this investigation suggest that renin, angiotensin II and ADH do not have significant influences on the decreased urine output. However, the decrease in alpha-ANP which might have been caused by the compression of the atria and the decrease of venous return induced by IPPV with PEEP, may be the cause of the decrease in urine output. Topics: Angiotensin II; Animals; Atrial Natriuretic Factor; Dogs; Intermittent Positive-Pressure Ventilation; Positive-Pressure Respiration; Renin; Urinary Retention; Vasopressins | 1990 |