pimagedine has been researched along with Brain Damage, Chronic in 3 studies
pimagedine: diamine oxidase & nitric oxide synthase inhibitor; an advanced glycosylation end product inhibitor; used in the treatment of diabetic complications; structure
aminoguanidine : A one-carbon compound whose unique structure renders it capable of acting as a derivative of hydrazine, guanidine or formamide.
Brain Damage, Chronic: A condition characterized by long-standing brain dysfunction or damage, usually of three months duration or longer. Potential etiologies include BRAIN INFARCTION; certain NEURODEGENERATIVE DISORDERS; CRANIOCEREBRAL TRAUMA; ANOXIA, BRAIN; ENCEPHALITIS; certain NEUROTOXICITY SYNDROMES; metabolic disorders (see BRAIN DISEASES, METABOLIC); and other conditions.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (66.67) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Zhang, F | 1 |
| Casey, RM | 1 |
| Ross, ME | 1 |
| Iadecola, C | 1 |
| Sinz, EH | 1 |
| Kochanek, PM | 1 |
| Dixon, CE | 1 |
| Clark, RS | 1 |
| Carcillo, JA | 1 |
| Schiding, JK | 1 |
| Chen, M | 1 |
| Wisniewski, SR | 1 |
| Carlos, TM | 1 |
| Williams, D | 1 |
| DeKosky, ST | 1 |
| Watkins, SC | 1 |
| Marion, DW | 1 |
| Billiar, TR | 1 |
| Stoffel, M | 1 |
| Rinecker, M | 1 |
| Plesnila, N | 1 |
| Eriskat, J | 1 |
| Baethmann, A | 1 |
3 other studies available for pimagedine and Brain Damage, Chronic
| Article | Year |
|---|---|
Aminoguanidine ameliorates and L-arginine worsens brain damage from intraluminal middle cerebral artery occlusion.
Topics: Animals; Arginine; Arterial Occlusive Diseases; Base Sequence; Body Water; Brain; Brain Damage, Chro | 1996 |
Inducible nitric oxide synthase is an endogenous neuroprotectant after traumatic brain injury in rats and mice.
Topics: Animals; Brain Damage, Chronic; Brain Injuries; Cognition Disorders; Enzyme Induction; Enzyme Inhibi | 1999 |
Attenuation of secondary lesion growth in the brain after trauma by selective inhibition of the inducible NO-synthase.
Topics: Animals; Brain Damage, Chronic; Brain Edema; Brain Injuries; Cerebral Cortex; Enzyme Inhibitors; Gua | 2000 |