pilocarpine has been researched along with Pulpitis in 3 studies
Pilocarpine: A slowly hydrolyzed muscarinic agonist with no nicotinic effects. Pilocarpine is used as a miotic and in the treatment of glaucoma.
(+)-pilocarpine : The (+)-enantiomer of pilocarpine.
Pulpitis: Inflammation of the DENTAL PULP, usually due to bacterial infection in dental caries, tooth fracture, or other conditions causing exposure of the pulp to bacterial invasion. Chemical irritants, thermal factors, hyperemic changes, and other factors may also cause pulpitis.
Excerpt | Relevance | Reference |
---|---|---|
"Pulpitis was induced with bacterial lipolysaccharide in rat incisors dental pulp." | 1.37 | Inflammation triggers constitutive activity and agonist-induced negative responses at M(3) muscarinic receptor in dental pulp. ( Borda, E; De Couto Pita, A; Orman, B; Sterin-Borda, L, 2011) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (66.67) | 29.6817 |
2010's | 1 (33.33) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
De Couto Pita, A | 3 |
Borda, E | 3 |
Ganzinelli, S | 2 |
Passafaro, D | 2 |
Sterin-Borda, L | 3 |
Orman, B | 1 |
3 other studies available for pilocarpine and Pulpitis
Article | Year |
---|---|
Cholinoceptor modulation on nitric oxide regulates prostaglandin E(2) and metalloproteinase-3 production in experimentally induced inflammation of rat dental pulp.
Topics: Animals; Cyclooxygenase 2; Dinoprostone; Inflammation Mediators; Male; Matrix Metalloproteinase 3; M | 2009 |
Differential cholinoceptor modulation of nitric oxide isoforms in experimentally-induced inflammation of dental pulp tissue.
Topics: Analysis of Variance; Animals; Dental Pulp; Male; Muscarinic Agonists; Nitric Oxide; Nitric Oxide Sy | 2009 |
Inflammation triggers constitutive activity and agonist-induced negative responses at M(3) muscarinic receptor in dental pulp.
Topics: Analysis of Variance; Animals; Dental Pulp; Disease Models, Animal; Drug Inverse Agonism; Inflammati | 2011 |