picrotoxin has been researched along with Metabolic Acidosis in 5 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (20.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (60.00) | 29.6817 |
| 2010's | 1 (20.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Albert, GW; Howard, MA; Schnizler, MK; Severson, MA; Welsh, MJ; Wemmie, JA; Ziemann, AE | 1 |
| Dong, Q; Liu, K; Liu, L; Liu, X; Su, J; Tang, M; Yang, X; Zhang, R; Zhou, H | 1 |
| Cao, H; Chu, XP; Duan, B; Hansen, J; Huang, Y; Simon, RP; Wang, YZ; Xiong, ZG; Xu, TL; Yang, T; Yu, Y; Zhu, MX | 1 |
| Chesler, M; Tong, CK | 1 |
| Bickerman, HA; Chusid, EL | 1 |
5 other study(ies) available for picrotoxin and Metabolic Acidosis
| Article | Year |
|---|---|
Seizure termination by acidosis depends on ASIC1a.
Topics: Acid Sensing Ion Channels; Acidosis; Analysis of Variance; Animals; Animals, Newborn; Behavior, Animal; Carbon Dioxide; Electroencephalography; Female; Hippocampus; Hydrogen-Ion Concentration; Interneurons; Kainic Acid; Male; Membrane Potentials; Mice; Mice, Knockout; Nerve Tissue Proteins; Patch-Clamp Techniques; Pentylenetetrazole; Picrotoxin; Seizures; Sodium Channels; Time Factors | 2008 |
Tissue kallikrein protects cortical neurons against in vitro ischemia-acidosis/reperfusion-induced injury through the ERK1/2 pathway.
Topics: Acid Sensing Ion Channels; Acidosis; Analysis of Variance; Animals; Animals, Newborn; Caspase 3; Cell Survival; Cells, Cultured; Central Nervous System Stimulants; Cerebral Cortex; Dose-Response Relationship, Drug; Enzyme Inhibitors; Glucose; Humans; Hydrogen-Ion Concentration; Hypoxia; In Situ Nick-End Labeling; L-Lactate Dehydrogenase; Membrane Potential, Mitochondrial; Microtubule-Associated Proteins; Mitogen-Activated Protein Kinase 3; Nerve Tissue Proteins; Neurons; Neuroprotective Agents; Picrotoxin; Rats; Rats, Sprague-Dawley; Reactive Oxygen Species; Sodium Channels; Tissue Kallikreins; Urine | 2009 |
Extracellular spermine exacerbates ischemic neuronal injury through sensitization of ASIC1a channels to extracellular acidosis.
Topics: Acid Sensing Ion Channels; Acidosis; Amino Acid Transport System y+; Animals; Biophysics; Brain Injuries; Calcium; Cells, Cultured; CHO Cells; Cricetinae; Cricetulus; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Combinations; Drug Interactions; Electric Stimulation; Embryo, Mammalian; Excitatory Amino Acid Antagonists; Extracellular Fluid; GABA Antagonists; Glucose; Hippocampus; Hydrogen-Ion Concentration; Hypoxia; Infarction, Middle Cerebral Artery; L-Lactate Dehydrogenase; Membrane Potentials; Mice; Mice, Knockout; Mutation; Nerve Tissue Proteins; Neurons; Oligonucleotides; Patch-Clamp Techniques; Picrotoxin; Putrescine; Sodium Channels; Spermine; Tetrazolium Salts; Time Factors; Transfection; Valine | 2011 |
Modulation of spreading depression by changes in extracellular pH.
Topics: Acidosis; Alkalosis; Animals; Benzolamide; Bicarbonates; Calcium; Carbon Dioxide; Carbonic Anhydrase Inhibitors; Convulsants; Cortical Spreading Depression; Extracellular Space; Hippocampus; Hydrogen-Ion Concentration; Membrane Potentials; Organ Culture Techniques; Picrotoxin; Rats; Rats, Sprague-Dawley; Receptors, N-Methyl-D-Aspartate | 2000 |
The case against the use of respiratory stimulants.
Topics: Acidosis; Benzoates; Blood Gas Analysis; Catechols; Central Nervous System Stimulants; Humans; Hypercapnia; Hyperventilation; Hypoxia; Lung Diseases; Nikethamide; Picrotoxin; Respiratory Insufficiency; Ventilation-Perfusion Ratio | 1970 |