physcione has been researched along with Liver Neoplasms in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (75.00) | 24.3611 |
| 2020's | 1 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Alanazy, M; Dewdney, B; George, J; Gillman, R; Hebbard, L; Qiao, L; Roberts, A; Walker, S; Wankell, M | 1 |
| Huang, LH; Li, Y; Pan, XP; Wang, C | 1 |
| Li, Y; Pan, X; Wang, C; Zhang, T; Zhu, L | 1 |
| Guo, P; Qin, F; Wang, Q; Wang, Y; Xing, Y; Yan, Y; Zhang, J; Zhuang, J | 1 |
4 other study(ies) available for physcione and Liver Neoplasms
| Article | Year |
|---|---|
The effects of fructose and metabolic inhibition on hepatocellular carcinoma.
Topics: Animals; Apoptosis; Carcinoma, Hepatocellular; Cell Line, Tumor; Cell Proliferation; Databases, Factual; Emodin; Energy Metabolism; Fructose; Humans; Liver Neoplasms; Mice; Survival Rate | 2020 |
Physcion induces apoptosis through triggering endoplasmic reticulum stress in hepatocellular carcinoma.
Topics: AMP-Activated Protein Kinases; Animals; Antineoplastic Agents, Phytogenic; Apoptosis; Carcinoma, Hepatocellular; Cell Line, Tumor; Emodin; Endoplasmic Reticulum Stress; Humans; Liver Neoplasms; Male; Mice; Mice, Inbred BALB C; Mice, Nude; Signal Transduction; Xenograft Model Antitumor Assays | 2018 |
RETRACTED: Protective autophagy induced by physcion suppresses hepatocellular carcinoma cell metastasis by inactivating the JAK2/STAT3 Axis.
Topics: Animals; Apoptosis; Autophagy; Carcinoma, Hepatocellular; Cell Line, Tumor; Cell Movement; Emodin; Humans; Janus Kinase 2; Liver Neoplasms; Male; Mice, Inbred BALB C; STAT3 Transcription Factor; Xenograft Model Antitumor Assays | 2018 |
Physcion 8-O-β-glucopyranoside induces apoptosis, suppresses invasion and inhibits epithelial to mesenchymal transition of hepatocellular carcinoma HepG2 cells.
Topics: Adenylate Kinase; Apoptosis; Carcinoma, Hepatocellular; Caspases; Cell Cycle Checkpoints; Cell Movement; Cell Proliferation; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; Down-Regulation; Emodin; Epithelial-Mesenchymal Transition; Glucosides; Hep G2 Cells; Humans; Liver Neoplasms; Neoplasm Invasiveness; Reactive Oxygen Species; Signal Transduction; Sp1 Transcription Factor | 2016 |