phosphotyrosine has been researched along with Myeloproliferative Disorders in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 2 (50.00) | 24.3611 |
| 2020's | 1 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Babon, JJ; Ellyard, JI; Kershaw, NJ; Laktyushin, A; Morris, R; Murphy, JM; Vinuesa, CG; Zhang, Y | 1 |
| Dagger, SA; Duyvestyn, JM; Langdon, WY; Morse, HC; Orandle, M; Taylor, SJ; Thien, CB | 1 |
| Elabd, M; Freund, P; Gouilleux, F; Groner, B; Hager, M; Han, X; Kerenyi, MA; Krämer, OH; Moriggl, R; Pham, HTT; Sexl, V; Valent, P; Wagner, T; Wingelhofer, B | 1 |
| Cheng, D; McLaughlin, J; Shokat, KM; Witte, ON; Wong, S; Zhang, C | 1 |
4 other study(ies) available for phosphotyrosine and Myeloproliferative Disorders
| Article | Year |
|---|---|
Structural and functional analysis of target recognition by the lymphocyte adaptor protein LNK.
Topics: Adaptor Proteins, Signal Transducing; Amino Acid Motifs; Animals; Binding Sites; Crystallography, X-Ray; fms-Like Tyrosine Kinase 3; Humans; Janus Kinase 2; Janus Kinase 3; Mice; Mutation; Myeloproliferative Disorders; Phosphotyrosine; Protein Binding; Proto-Oncogene Proteins c-kit; Receptors, Erythropoietin; Signal Transduction; src Homology Domains | 2021 |
Dasatinib targets B-lineage cells but does not provide an effective therapy for myeloproliferative disease in c-Cbl RING finger mutant mice.
Topics: Animals; B-Lymphocytes; Bone Marrow Cells; Cell Lineage; Dasatinib; Dose-Response Relationship, Drug; Germinal Center; Hematopoietic Stem Cells; Humans; Lymphocyte Count; Male; Mice, Inbred C57BL; Mice, Mutant Strains; Myeloproliferative Disorders; Neutrophils; Phosphorylation; Phosphotyrosine; Precursor Cells, B-Lymphoid; Proto-Oncogene Proteins c-cbl; Pyrimidines; RING Finger Domains; src-Family Kinases; Thiazoles | 2014 |
O-GlcNAcylation of STAT5 controls tyrosine phosphorylation and oncogenic transcription in STAT5-dependent malignancies.
Topics: Acetylglucosamine; Animals; Cell Line; Cell Transformation, Neoplastic; Female; Gene Expression Regulation, Neoplastic; Genes, Reporter; Glycosylation; Humans; Interleukin-3; Lymphoid Tissue; Male; Mice; Mutagenesis, Site-Directed; Myeloproliferative Disorders; Phosphorylation; Phosphotyrosine; Protein Processing, Post-Translational; Radiation Chimera; Recombinant Fusion Proteins; Signal Transduction; STAT5 Transcription Factor; T-Lymphocytes; Threonine; Transcriptional Activation; Tumor Suppressor Proteins | 2017 |
Sole BCR-ABL inhibition is insufficient to eliminate all myeloproliferative disorder cell populations.
Topics: Apoptosis; B-Lymphocytes; Benzamides; Cell Cycle; Cell Line; Cell Proliferation; Drug Synergism; Enzyme Inhibitors; Fusion Proteins, bcr-abl; Gene Expression Regulation, Enzymologic; Humans; Imatinib Mesylate; Mutation; Myeloproliferative Disorders; Phosphotyrosine; Piperazines; Protein-Tyrosine Kinases; Proto-Oncogene Proteins c-kit; Pyrimidines; Tyrosine | 2004 |