Page last updated: 2024-08-23

phosphotyrosine and Mast-Cell Leukemia

phosphotyrosine has been researched along with Mast-Cell Leukemia in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	1 (50.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Buet, D; Denis, N; Dusanter-Fourt, I; Gallais, I; Guillonneau, F; Guillouf, C; Kosmider, O; Lauret, E; Loew, D; Lombard, B; Mayeux, P; Moreau-Gachelin, F	1
Ashman, LK; Butterfield, JH; Furitsu, T; Ikeda, H; Kanayama, Y; Kitayama, H; Koshimizu, U; Sugahara, H; Tono, T; Tsujimura, T	1

Other Studies

2 other study(ies) available for phosphotyrosine and Mast-Cell Leukemia

Article	Year
Cotargeting signaling pathways driving survival and cell cycle circumvents resistance to Kit inhibitors in leukemia. Blood, 2012, May-03, Volume: 119, Issue:18 Topics: Adenylate Kinase; Animals; Antineoplastic Agents; Cell Cycle; Cell Line, Tumor; Cell Survival; Drug Resistance, Neoplasm; Female; Humans; Imidazoles; Indoles; Leukemia, Mast-Cell; Mice; Mice, Nude; Mice, Transgenic; Neoplasm Proteins; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Phosphorylation; Phosphotyrosine; Protein Kinase Inhibitors; Protein Processing, Post-Translational; Protein Tyrosine Phosphatase, Non-Receptor Type 11; Proto-Oncogene Proteins c-kit; Pyrroles; Quinolines; RNA Interference; RNA, Small Interfering; Signal Transduction; STAT5 Transcription Factor; Tumor Stem Cell Assay	2012
Identification of mutations in the coding sequence of the proto-oncogene c-kit in a human mast cell leukemia cell line causing ligand-independent activation of c-kit product. The Journal of clinical investigation, 1993, Volume: 92, Issue:4 Topics: Amino Acid Sequence; Animals; Base Sequence; Cell Line; DNA Primers; Granulocyte-Macrophage Colony-Stimulating Factor; Humans; Interleukin-3; Leukemia, Mast-Cell; Mice; Molecular Sequence Data; Phosphotyrosine; Point Mutation; Polymerase Chain Reaction; Proto-Oncogene Mas; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-kit; Proto-Oncogenes; Receptor Protein-Tyrosine Kinases; Receptors, Colony-Stimulating Factor; Recombinant Proteins; Transfection; Tumor Cells, Cultured; Tyrosine	1993

Article

Year

Cotargeting signaling pathways driving survival and cell cycle circumvents resistance to Kit inhibitors in leukemia.

Blood, 2012, May-03, Volume: 119, Issue:18

Topics: Adenylate Kinase; Animals; Antineoplastic Agents; Cell Cycle; Cell Line, Tumor; Cell Survival; Drug Resistance, Neoplasm; Female; Humans; Imidazoles; Indoles; Leukemia, Mast-Cell; Mice; Mice, Nude; Mice, Transgenic; Neoplasm Proteins; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Phosphorylation; Phosphotyrosine; Protein Kinase Inhibitors; Protein Processing, Post-Translational; Protein Tyrosine Phosphatase, Non-Receptor Type 11; Proto-Oncogene Proteins c-kit; Pyrroles; Quinolines; RNA Interference; RNA, Small Interfering; Signal Transduction; STAT5 Transcription Factor; Tumor Stem Cell Assay

2012

Identification of mutations in the coding sequence of the proto-oncogene c-kit in a human mast cell leukemia cell line causing ligand-independent activation of c-kit product.

The Journal of clinical investigation, 1993, Volume: 92, Issue:4

Topics: Amino Acid Sequence; Animals; Base Sequence; Cell Line; DNA Primers; Granulocyte-Macrophage Colony-Stimulating Factor; Humans; Interleukin-3; Leukemia, Mast-Cell; Mice; Molecular Sequence Data; Phosphotyrosine; Point Mutation; Polymerase Chain Reaction; Proto-Oncogene Mas; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-kit; Proto-Oncogenes; Receptor Protein-Tyrosine Kinases; Receptors, Colony-Stimulating Factor; Recombinant Proteins; Transfection; Tumor Cells, Cultured; Tyrosine

1993