Compounds > phosphotyrosine
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phosphotyrosine and Cancer of Gastrointestinal Tract

phosphotyrosine has been researched along with Cancer of Gastrointestinal Tract in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (33.33)18.2507
2000's1 (33.33)29.6817
2010's1 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Dent, P; Grant, S; Häussinger, D; Ogretmen, B; Park, MA; Reinehr, R; Voelkel-Johnson, C; Yacoub, A1
Azuma, T; Hatakeyama, M; Higashi, H; Iwabuchi, K; Matsui, A; Miura, M; Musashi, M; Ohnishi, N; Sawa, H; Suzuki, M; Tanaka, S; Yamada, G; Yuasa, H1
Hanada, M; Hashimoto, K; Hirota, S; Ishiguro, S; Isozaki, K; Kanakura, Y; Kawano, K; Kitamura, Y; Kurata, A; Matsuzawa, Y; Moriyama, Y; Muhammad Tunio, G; Nishida, T; Shinomura, Y; Takeda, M1

Other Studies

3 other study(ies) available for phosphotyrosine and Cancer of Gastrointestinal Tract

ArticleYear
Sorafenib activates CD95 and promotes autophagy and cell death via Src family kinases in gastrointestinal tumor cells.
    Molecular cancer therapeutics, 2010, Volume: 9, Issue:8

    Topics: Autophagy; Benzenesulfonates; Cell Line, Tumor; Dose-Response Relationship, Drug; Enzyme Activation; Fas Ligand Protein; fas Receptor; Gastrointestinal Neoplasms; Humans; Hydroxamic Acids; Niacinamide; Phenylurea Compounds; Phosphotyrosine; Protein Kinase Inhibitors; Pyridines; Reactive Oxygen Species; Receptor, Platelet-Derived Growth Factor beta; Signal Transduction; Sorafenib; src-Family Kinases; Vorinostat

2010
Transgenic expression of Helicobacter pylori CagA induces gastrointestinal and hematopoietic neoplasms in mouse.
    Proceedings of the National Academy of Sciences of the United States of America, 2008, Jan-22, Volume: 105, Issue:3

    Topics: Animals; Antigens, Bacterial; Bacterial Proteins; Cell Transformation, Neoplastic; Gastrointestinal Neoplasms; Gene Expression Regulation, Neoplastic; Helicobacter pylori; Hematologic Neoplasms; Mice; Mice, Transgenic; Phosphotyrosine

2008
Gain-of-function mutations of c-kit in human gastrointestinal stromal tumors.
    Science (New York, N.Y.), 1998, Jan-23, Volume: 279, Issue:5350

    Topics: Amino Acid Sequence; Animals; Antigens, CD34; Cell Line; Cell Transformation, Neoplastic; Digestive System; DNA, Complementary; Esophageal Neoplasms; Gastrointestinal Neoplasms; Humans; Intestinal Neoplasms; Ligands; Mice; Mice, Nude; Molecular Sequence Data; Mutation; Phosphorylation; Phosphotyrosine; Proto-Oncogene Proteins c-kit; Recombinant Proteins; Sequence Deletion; Stem Cell Factor; Stomach Neoplasms; Transfection

1998