phosphoserine has been researched along with Status Epilepticus in 3 studies
Phosphoserine: The phosphoric acid ester of serine.
Status Epilepticus: A prolonged seizure or seizures repeated frequently enough to prevent recovery between episodes occurring over a period of 20-30 minutes. The most common subtype is generalized tonic-clonic status epilepticus, a potentially fatal condition associated with neuronal injury and respiratory and metabolic dysfunction. Nonconvulsive forms include petit mal status and complex partial status, which may manifest as behavioral disturbances. Simple partial status epilepticus consists of persistent motor, sensory, or autonomic seizures that do not impair cognition (see also EPILEPSIA PARTIALIS CONTINUA). Subclinical status epilepticus generally refers to seizures occurring in an unresponsive or comatose individual in the absence of overt signs of seizure activity. (From N Engl J Med 1998 Apr 2;338(14):970-6; Neurologia 1997 Dec;12 Suppl 6:25-30)
Excerpt | Relevance | Reference |
---|---|---|
"Recently, several groups reported that seizures increase mammalian target of rapamycin (mTOR) kinase activity, and such increased activity in genetic models can contribute to spontaneous seizures." | 5.39 | Spatiotemporal characterization of mTOR kinase activity following kainic acid induced status epilepticus and analysis of rat brain response to chronic rapamycin treatment. ( Blazejczyk, M; Caban, B; Jaworski, J; Kazmierska, P; Konopacki, J; Macias, M; Rodo, A; Skalecka, A; Tarkowski, B, 2013) |
"Recently, several groups reported that seizures increase mammalian target of rapamycin (mTOR) kinase activity, and such increased activity in genetic models can contribute to spontaneous seizures." | 1.39 | Spatiotemporal characterization of mTOR kinase activity following kainic acid induced status epilepticus and analysis of rat brain response to chronic rapamycin treatment. ( Blazejczyk, M; Caban, B; Jaworski, J; Kazmierska, P; Konopacki, J; Macias, M; Rodo, A; Skalecka, A; Tarkowski, B, 2013) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (66.67) | 24.3611 |
2020's | 1 (33.33) | 2.80 |
Authors | Studies |
---|---|
Kim, JE | 1 |
Park, H | 1 |
Lee, JE | 1 |
Kang, TC | 1 |
Macias, M | 1 |
Blazejczyk, M | 1 |
Kazmierska, P | 1 |
Caban, B | 1 |
Skalecka, A | 1 |
Tarkowski, B | 1 |
Rodo, A | 1 |
Konopacki, J | 1 |
Jaworski, J | 1 |
Silayeva, L | 1 |
Deeb, TZ | 1 |
Hines, RM | 1 |
Kelley, MR | 1 |
Munoz, MB | 1 |
Lee, HH | 1 |
Brandon, NJ | 1 |
Dunlop, J | 1 |
Maguire, J | 1 |
Davies, PA | 1 |
Moss, SJ | 1 |
3 other studies available for phosphoserine and Status Epilepticus
Article | Year |
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CDDO-Me Inhibits Microglial Activation and Monocyte Infiltration by Abrogating NFκB- and p38 MAPK-Mediated Signaling Pathways Following Status Epilepticus.
Topics: Animals; Chemokine CCL2; Frontal Lobe; Male; MAP Kinase Signaling System; Microglia; Models, Biologi | 2020 |
Spatiotemporal characterization of mTOR kinase activity following kainic acid induced status epilepticus and analysis of rat brain response to chronic rapamycin treatment.
Topics: Animals; Astrocytes; Brain; Cell Death; Cell Nucleus; Hippocampus; Kainic Acid; Male; Neurons; Phosp | 2013 |
KCC2 activity is critical in limiting the onset and severity of status epilepticus.
Topics: Animals; Chlorides; Endocytosis; gamma-Aminobutyric Acid; Gene Knock-In Techniques; Glutamates; K Cl | 2015 |