phosphoserine has been researched along with Absence Status in 3 studies
Phosphoserine: The phosphoric acid ester of serine.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Recently, several groups reported that seizures increase mammalian target of rapamycin (mTOR) kinase activity, and such increased activity in genetic models can contribute to spontaneous seizures." | 5.39 | Spatiotemporal characterization of mTOR kinase activity following kainic acid induced status epilepticus and analysis of rat brain response to chronic rapamycin treatment. ( Blazejczyk, M; Caban, B; Jaworski, J; Kazmierska, P; Konopacki, J; Macias, M; Rodo, A; Skalecka, A; Tarkowski, B, 2013) |
| "Recently, several groups reported that seizures increase mammalian target of rapamycin (mTOR) kinase activity, and such increased activity in genetic models can contribute to spontaneous seizures." | 1.39 | Spatiotemporal characterization of mTOR kinase activity following kainic acid induced status epilepticus and analysis of rat brain response to chronic rapamycin treatment. ( Blazejczyk, M; Caban, B; Jaworski, J; Kazmierska, P; Konopacki, J; Macias, M; Rodo, A; Skalecka, A; Tarkowski, B, 2013) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 1 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Kim, JE | 1 |
| Park, H | 1 |
| Lee, JE | 1 |
| Kang, TC | 1 |
| Macias, M | 1 |
| Blazejczyk, M | 1 |
| Kazmierska, P | 1 |
| Caban, B | 1 |
| Skalecka, A | 1 |
| Tarkowski, B | 1 |
| Rodo, A | 1 |
| Konopacki, J | 1 |
| Jaworski, J | 1 |
| Silayeva, L | 1 |
| Deeb, TZ | 1 |
| Hines, RM | 1 |
| Kelley, MR | 1 |
| Munoz, MB | 1 |
| Lee, HH | 1 |
| Brandon, NJ | 1 |
| Dunlop, J | 1 |
| Maguire, J | 1 |
| Davies, PA | 1 |
| Moss, SJ | 1 |
3 other studies available for phosphoserine and Absence Status
| Article | Year |
|---|---|
CDDO-Me Inhibits Microglial Activation and Monocyte Infiltration by Abrogating NFκB- and p38 MAPK-Mediated Signaling Pathways Following Status Epilepticus.
Topics: Animals; Chemokine CCL2; Frontal Lobe; Male; MAP Kinase Signaling System; Microglia; Models, Biologi | 2020 |
Spatiotemporal characterization of mTOR kinase activity following kainic acid induced status epilepticus and analysis of rat brain response to chronic rapamycin treatment.
Topics: Animals; Astrocytes; Brain; Cell Death; Cell Nucleus; Hippocampus; Kainic Acid; Male; Neurons; Phosp | 2013 |
KCC2 activity is critical in limiting the onset and severity of status epilepticus.
Topics: Animals; Chlorides; Endocytosis; gamma-Aminobutyric Acid; Gene Knock-In Techniques; Glutamates; K Cl | 2015 |