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phosphorylethanolamine and Seizures

phosphorylethanolamine has been researched along with Seizures in 4 studies

phosphorylethanolamine: RN given refers to parent cpd; structure
O-phosphoethanolamine : The ethanolamine mono-ester of phosphoric acid, and a metabolite of phospholipid metabolism. This phosphomonoester shows strong structural similarity to the inhibitory neurotransmitter GABA, and is decreased in post-mortem Alzheimer's disease brain.

Seizures: Clinical or subclinical disturbances of cortical function due to a sudden, abnormal, excessive, and disorganized discharge of brain cells. Clinical manifestations include abnormal motor, sensory and psychic phenomena. Recurrent seizures are usually referred to as EPILEPSY or seizure disorder.

Research Excerpts

ExcerptRelevanceReference
"Pyridoxine-responsive seizures (PRS) and the role of pyridoxine (PN, vitamin B(6)) in hypophosphatasia (HPP) are incompletely understood."7.74Pyridoxine-responsive seizures as the first symptom of infantile hypophosphatasia caused by two novel missense mutations (c.677T>C, p.M226T; c.1112C>T, p.T371I) of the tissue-nonspecific alkaline phosphatase gene. ( Baumgartner-Sigl, S; Ericson, KL; Haberlandt, E; Högler, W; Mumm, S; Ryan, L; Scholl-Bürgi, S; Sergi, C; Whyte, MP, 2007)
"The incorporation of labeled ethanolamine into phosphatidylethanolamine (PE) and its water-soluble precursors, phosphoethanolamine and CDP-ethanolamine, is measured in rat cerebella during the course of bicuculline-induced convulsive seizures."7.67Cerebellar metabolism of phosphatidylethanolamine and its water-soluble precursors during bicuculline-induced convulsive seizures. ( Arienti, G; Corazzi, L; Marku, N; Piccinin, GL, 1987)
"Pyridoxine-responsive seizures (PRS) and the role of pyridoxine (PN, vitamin B(6)) in hypophosphatasia (HPP) are incompletely understood."3.74Pyridoxine-responsive seizures as the first symptom of infantile hypophosphatasia caused by two novel missense mutations (c.677T>C, p.M226T; c.1112C>T, p.T371I) of the tissue-nonspecific alkaline phosphatase gene. ( Baumgartner-Sigl, S; Ericson, KL; Haberlandt, E; Högler, W; Mumm, S; Ryan, L; Scholl-Bürgi, S; Sergi, C; Whyte, MP, 2007)
"The incorporation of labeled ethanolamine into phosphatidylethanolamine (PE) and its water-soluble precursors, phosphoethanolamine and CDP-ethanolamine, is measured in rat cerebella during the course of bicuculline-induced convulsive seizures."3.67Cerebellar metabolism of phosphatidylethanolamine and its water-soluble precursors during bicuculline-induced convulsive seizures. ( Arienti, G; Corazzi, L; Marku, N; Piccinin, GL, 1987)
"Pyridoxine-sensitive seizures characterize severe forms of infantile HPP."1.46Neuromuscular features of hypophosphatasia. ( Fonta, C; Salles, JP, 2017)
"Hypophosphatasia is a rare genetic disease characterized by deficiency of tissue-nonspecific alkaline phosphatase (TNSALP) activity, excessive urinary excretion of phosphoethanolamine, poor bone mineralization and skeletal anomalies."1.33Neonatal hypophosphatasia and seizures. A case report. ( Meli, C; Palazzo, P; Romeo, DM; Smilari, P; Sorge, G, 2005)

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19901 (25.00)18.7374
1990's0 (0.00)18.2507
2000's2 (50.00)29.6817
2010's1 (25.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Fonta, C1
Salles, JP1
Smilari, P1
Romeo, DM1
Palazzo, P1
Meli, C1
Sorge, G1
Baumgartner-Sigl, S1
Haberlandt, E1
Mumm, S1
Scholl-Bürgi, S1
Sergi, C1
Ryan, L1
Ericson, KL1
Whyte, MP1
Högler, W1
Marku, N1
Corazzi, L1
Piccinin, GL1
Arienti, G1

Other Studies

4 other studies available for phosphorylethanolamine and Seizures

ArticleYear
Neuromuscular features of hypophosphatasia.
    Archives de pediatrie : organe officiel de la Societe francaise de pediatrie, 2017, Volume: 24, Issue:5S2

    Topics: Adult; Alkaline Phosphatase; Animals; Biomarkers; Brain Diseases; Chronic Pain; Disease Models, Anim

2017
Neonatal hypophosphatasia and seizures. A case report.
    Minerva pediatrica, 2005, Volume: 57, Issue:5

    Topics: Chromatography, Ion Exchange; Ethanolamines; Humans; Hypophosphatasia; Infant; Infant, Newborn; Infa

2005
Pyridoxine-responsive seizures as the first symptom of infantile hypophosphatasia caused by two novel missense mutations (c.677T>C, p.M226T; c.1112C>T, p.T371I) of the tissue-nonspecific alkaline phosphatase gene.
    Bone, 2007, Volume: 40, Issue:6

    Topics: Alkaline Phosphatase; Ethanolamines; Exons; Fatal Outcome; Female; Humans; Hypercalcemia; Hypercalci

2007
Cerebellar metabolism of phosphatidylethanolamine and its water-soluble precursors during bicuculline-induced convulsive seizures.
    Neurochemical research, 1987, Volume: 12, Issue:4

    Topics: Animals; Bicuculline; Cerebellum; Cytidine Diphosphate; Ethanolamines; Female; Phosphatidylethanolam

1987