phosphorylethanolamine has been researched along with Seizures in 4 studies
phosphorylethanolamine: RN given refers to parent cpd; structure
O-phosphoethanolamine : The ethanolamine mono-ester of phosphoric acid, and a metabolite of phospholipid metabolism. This phosphomonoester shows strong structural similarity to the inhibitory neurotransmitter GABA, and is decreased in post-mortem Alzheimer's disease brain.
Seizures: Clinical or subclinical disturbances of cortical function due to a sudden, abnormal, excessive, and disorganized discharge of brain cells. Clinical manifestations include abnormal motor, sensory and psychic phenomena. Recurrent seizures are usually referred to as EPILEPSY or seizure disorder.
Excerpt | Relevance | Reference |
---|---|---|
"Pyridoxine-responsive seizures (PRS) and the role of pyridoxine (PN, vitamin B(6)) in hypophosphatasia (HPP) are incompletely understood." | 7.74 | Pyridoxine-responsive seizures as the first symptom of infantile hypophosphatasia caused by two novel missense mutations (c.677T>C, p.M226T; c.1112C>T, p.T371I) of the tissue-nonspecific alkaline phosphatase gene. ( Baumgartner-Sigl, S; Ericson, KL; Haberlandt, E; Högler, W; Mumm, S; Ryan, L; Scholl-Bürgi, S; Sergi, C; Whyte, MP, 2007) |
"The incorporation of labeled ethanolamine into phosphatidylethanolamine (PE) and its water-soluble precursors, phosphoethanolamine and CDP-ethanolamine, is measured in rat cerebella during the course of bicuculline-induced convulsive seizures." | 7.67 | Cerebellar metabolism of phosphatidylethanolamine and its water-soluble precursors during bicuculline-induced convulsive seizures. ( Arienti, G; Corazzi, L; Marku, N; Piccinin, GL, 1987) |
"Pyridoxine-responsive seizures (PRS) and the role of pyridoxine (PN, vitamin B(6)) in hypophosphatasia (HPP) are incompletely understood." | 3.74 | Pyridoxine-responsive seizures as the first symptom of infantile hypophosphatasia caused by two novel missense mutations (c.677T>C, p.M226T; c.1112C>T, p.T371I) of the tissue-nonspecific alkaline phosphatase gene. ( Baumgartner-Sigl, S; Ericson, KL; Haberlandt, E; Högler, W; Mumm, S; Ryan, L; Scholl-Bürgi, S; Sergi, C; Whyte, MP, 2007) |
"The incorporation of labeled ethanolamine into phosphatidylethanolamine (PE) and its water-soluble precursors, phosphoethanolamine and CDP-ethanolamine, is measured in rat cerebella during the course of bicuculline-induced convulsive seizures." | 3.67 | Cerebellar metabolism of phosphatidylethanolamine and its water-soluble precursors during bicuculline-induced convulsive seizures. ( Arienti, G; Corazzi, L; Marku, N; Piccinin, GL, 1987) |
"Pyridoxine-sensitive seizures characterize severe forms of infantile HPP." | 1.46 | Neuromuscular features of hypophosphatasia. ( Fonta, C; Salles, JP, 2017) |
"Hypophosphatasia is a rare genetic disease characterized by deficiency of tissue-nonspecific alkaline phosphatase (TNSALP) activity, excessive urinary excretion of phosphoethanolamine, poor bone mineralization and skeletal anomalies." | 1.33 | Neonatal hypophosphatasia and seizures. A case report. ( Meli, C; Palazzo, P; Romeo, DM; Smilari, P; Sorge, G, 2005) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 1 (25.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (50.00) | 29.6817 |
2010's | 1 (25.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Fonta, C | 1 |
Salles, JP | 1 |
Smilari, P | 1 |
Romeo, DM | 1 |
Palazzo, P | 1 |
Meli, C | 1 |
Sorge, G | 1 |
Baumgartner-Sigl, S | 1 |
Haberlandt, E | 1 |
Mumm, S | 1 |
Scholl-Bürgi, S | 1 |
Sergi, C | 1 |
Ryan, L | 1 |
Ericson, KL | 1 |
Whyte, MP | 1 |
Högler, W | 1 |
Marku, N | 1 |
Corazzi, L | 1 |
Piccinin, GL | 1 |
Arienti, G | 1 |
4 other studies available for phosphorylethanolamine and Seizures
Article | Year |
---|---|
Neuromuscular features of hypophosphatasia.
Topics: Adult; Alkaline Phosphatase; Animals; Biomarkers; Brain Diseases; Chronic Pain; Disease Models, Anim | 2017 |
Neonatal hypophosphatasia and seizures. A case report.
Topics: Chromatography, Ion Exchange; Ethanolamines; Humans; Hypophosphatasia; Infant; Infant, Newborn; Infa | 2005 |
Pyridoxine-responsive seizures as the first symptom of infantile hypophosphatasia caused by two novel missense mutations (c.677T>C, p.M226T; c.1112C>T, p.T371I) of the tissue-nonspecific alkaline phosphatase gene.
Topics: Alkaline Phosphatase; Ethanolamines; Exons; Fatal Outcome; Female; Humans; Hypercalcemia; Hypercalci | 2007 |
Cerebellar metabolism of phosphatidylethanolamine and its water-soluble precursors during bicuculline-induced convulsive seizures.
Topics: Animals; Bicuculline; Cerebellum; Cytidine Diphosphate; Ethanolamines; Female; Phosphatidylethanolam | 1987 |