Page last updated: 2024-10-20

phosphorylethanolamine and Hyperinsulinism

phosphorylethanolamine has been researched along with Hyperinsulinism in 1 studies

phosphorylethanolamine: RN given refers to parent cpd; structure
O-phosphoethanolamine : The ethanolamine mono-ester of phosphoric acid, and a metabolite of phospholipid metabolism. This phosphomonoester shows strong structural similarity to the inhibitory neurotransmitter GABA, and is decreased in post-mortem Alzheimer's disease brain.

Hyperinsulinism: A syndrome with excessively high INSULIN levels in the BLOOD. It may cause HYPOGLYCEMIA. Etiology of hyperinsulinism varies, including hypersecretion of a beta cell tumor (INSULINOMA); autoantibodies against insulin (INSULIN ANTIBODIES); defective insulin receptor (INSULIN RESISTANCE); or overuse of exogenous insulin or HYPOGLYCEMIC AGENTS.

Research Excerpts

ExcerptRelevanceReference
"Here, we established an in vitro hyperinsulinemia-induced IR model in the HepG2 human liver cancer cell line and primary mouse hepatocyte via a high dose of insulin treatment."1.91ETNPPL modulates hyperinsulinemia-induced insulin resistance through the SIK1/ROS-mediated inactivation of the PI3K/AKT signaling pathway in hepatocytes. ( Chen, X; Fan, X; Han, F; Huang, Y; Li, M; Li, W; Li, X; Li, Y; Liu, P; Liu, W; Qian, L; Sun, X; Wang, C; Xiong, Y; Zhang, W, 2023)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's1 (100.00)2.80

Authors

AuthorsStudies
Chen, X1
Liu, P1
Zhang, W1
Li, X1
Wang, C1
Han, F1
Liu, W1
Huang, Y1
Li, M1
Li, Y1
Sun, X1
Fan, X1
Li, W1
Xiong, Y1
Qian, L1

Other Studies

1 other study available for phosphorylethanolamine and Hyperinsulinism

ArticleYear
ETNPPL modulates hyperinsulinemia-induced insulin resistance through the SIK1/ROS-mediated inactivation of the PI3K/AKT signaling pathway in hepatocytes.
    Journal of cellular physiology, 2023, Volume: 238, Issue:5

    Topics: Animals; Diabetes Mellitus, Type 2; Hepatocytes; Humans; Hyperinsulinism; Insulin; Insulin Resistanc

2023