Compounds > phorbolol myristate acetate
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phorbolol myristate acetate and Fibrosarcoma

phorbolol myristate acetate has been researched along with Fibrosarcoma in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (33.33)18.2507
2000's0 (0.00)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Choi, YJ; Lee, ST; Lee, YH1
Choi, JH; Hwang, YP; Jeong, HG; Kang, KW; Yun, HJ1
Belizario, JE; Dinarello, CA1

Other Studies

3 other study(ies) available for phorbolol myristate acetate and Fibrosarcoma

ArticleYear
Galangin and kaempferol suppress phorbol-12-myristate-13-acetate-induced matrix metalloproteinase-9 expression in human fibrosarcoma HT-1080 cells.
    Molecules and cells, 2015, Volume: 38, Issue:2

    Topics: Cell Line, Tumor; Cell Proliferation; Cell Survival; Fibrosarcoma; Flavonoids; Gene Expression Regulation, Neoplastic; Humans; I-kappa B Proteins; Kaempferols; Matrix Metalloproteinase 9; NF-KappaB Inhibitor alpha; Phosphorylation; Signal Transduction; Tetradecanoylphorbol Acetate

2015
Suppression of phorbol-12-myristate-13-acetate-induced tumor cell invasion by bergamottin via the inhibition of protein kinase Cdelta/p38 mitogen-activated protein kinase and JNK/nuclear factor-kappaB-dependent matrix metalloproteinase-9 expression.
    Molecular nutrition & food research, 2010, Volume: 54, Issue:7

    Topics: Antineoplastic Agents, Phytogenic; Carcinogens; Cell Line, Tumor; Cell Movement; Cytochrome P-450 Enzyme Inhibitors; Down-Regulation; Fibrosarcoma; Furocoumarins; Humans; I-kappa B Proteins; JNK Mitogen-Activated Protein Kinases; Matrix Metalloproteinase 2; Matrix Metalloproteinase 9; Neoplasm Invasiveness; NF-kappa B; NF-KappaB Inhibitor alpha; Osmolar Concentration; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein Kinase C-delta; Protein Transport; RNA, Messenger; Signal Transduction; Tetradecanoylphorbol Acetate

2010
Interleukin 1, interleukin 6, tumor necrosis factor, and transforming growth factor beta increase cell resistance to tumor necrosis factor cytotoxicity by growth arrest in the G1 phase of the cell cycle.
    Cancer research, 1991, May-01, Volume: 51, Issue:9

    Topics: Animals; Cell Cycle; Cholera Toxin; Dactinomycin; Dinoprostone; Dose-Response Relationship, Drug; Drug Resistance; Drug Screening Assays, Antitumor; Drug Synergism; Fibrosarcoma; G1 Phase; Humans; Indomethacin; Interleukin-1; Interleukin-6; Mice; Premedication; Tetradecanoylphorbol Acetate; Time Factors; Transforming Growth Factor beta; Tumor Cells, Cultured; Tumor Necrosis Factor-alpha

1991