phenytoin has been researched along with Allergic Encephalomyelitis in 8 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 6 (75.00) | 29.6817 |
2010's | 2 (25.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Black, JA; Liu, S; Waxman, SG; Zwinger, P | 1 |
Araya, SI; Hashiba, N; Inada, H; Matsui, M; Nagayama, S; Sonobe, Y; Suzumura, A | 1 |
Black, JA; Lo, AC; Waxman, SG | 1 |
Black, JA; Lo, AC; Saab, CY; Waxman, SG | 1 |
Black, JA; Craner, MJ; Cuzner, ML; Damarjian, TG; Hains, BC; Liu, S; Lo, AC; Newcombe, J; Waxman, SG | 1 |
Black, JA; Hains, BC; Liu, S; Saab, CY; Waxman, SG | 1 |
Black, JA; Carrithers, LM; Carrithers, M; Liu, S; Waxman, SG | 1 |
Black, JA; Waxman, SG | 1 |
1 review(s) available for phenytoin and Allergic Encephalomyelitis
Article | Year |
---|---|
Phenytoin protects central axons in experimental autoimmune encephalomyelitis.
Topics: Animals; Axons; Central Nervous System; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Humans; Mice; Models, Biological; Phenytoin | 2008 |
7 other study(ies) available for phenytoin and Allergic Encephalomyelitis
Article | Year |
---|---|
Tapered withdrawal of phenytoin removes protective effect in EAE without inflammatory rebound and mortality.
Topics: Animals; Disease Models, Animal; Drug Administration Schedule; Encephalomyelitis, Autoimmune, Experimental; Mice; Mice, Inbred C57BL; Myelin-Oligodendrocyte Glycoprotein; Neurologic Examination; Neuroprotective Agents; Peptide Fragments; Phenytoin; Time Factors | 2014 |
Phenytoin at optimum doses ameliorates experimental autoimmune encephalomyelitis via modulation of immunoregulatory cells.
Topics: Animals; Anticonvulsants; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Immunologic Factors; Mice; Mice, Inbred C57BL; Phenytoin; T-Lymphocytes, Regulatory | 2011 |
Neuroprotection of axons with phenytoin in experimental allergic encephalomyelitis.
Topics: Animals; Axons; Cell Death; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Immunohistochemistry; Mice; Mice, Inbred C57BL; Myelin Basic Protein; Nerve Degeneration; Nerve Fibers, Myelinated; Neurofilament Proteins; Neuroprotective Agents; Optic Nerve; Phenytoin; Sodium Channel Blockers; Sodium Channels; Treatment Outcome | 2002 |
Phenytoin protects spinal cord axons and preserves axonal conduction and neurological function in a model of neuroinflammation in vivo.
Topics: Animals; Axonal Transport; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Male; Mice; Mice, Inbred C57BL; Neural Conduction; Phenytoin; Spinal Cord | 2003 |
Sodium channels contribute to microglia/macrophage activation and function in EAE and MS.
Topics: Animals; Axons; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Female; Gliosis; Macrophages; Male; Mice; Mice, Inbred C57BL; Microglia; Multiple Sclerosis; NAV1.6 Voltage-Gated Sodium Channel; Nerve Degeneration; Nerve Tissue Proteins; Neuroprotective Agents; Phagocytosis; Phenytoin; RNA, Messenger; Sodium Channel Blockers; Sodium Channels; Tetrodotoxin; Up-Regulation | 2005 |
Long-term protection of central axons with phenytoin in monophasic and chronic-relapsing EAE.
Topics: Action Potentials; Administration, Oral; Animals; Axons; Cell Count; Cervical Vertebrae; Chronic Disease; Encephalomyelitis, Autoimmune, Experimental; Immunohistochemistry; Injections, Subcutaneous; Mice; Mice, Inbred C57BL; Myelin Proteins; Myelin-Associated Glycoprotein; Myelin-Oligodendrocyte Glycoprotein; Neural Conduction; Phenytoin; Recurrence; Sodium Channel Blockers; Spinal Cord; Treatment Outcome | 2006 |
Exacerbation of experimental autoimmune encephalomyelitis after withdrawal of phenytoin and carbamazepine.
Topics: Animals; Anticonvulsants; Antigens, CD; Axons; Carbamazepine; Cell Count; Disease Models, Animal; Encephalomyelitis, Autoimmune, Experimental; Flow Cytometry; Gene Expression Regulation; Glycoproteins; Mice; Mice, Inbred C57BL; Myelin-Oligodendrocyte Glycoprotein; NAV1.6 Voltage-Gated Sodium Channel; Nerve Tissue Proteins; Peptide Fragments; Phenytoin; Pyramidal Tracts; Severity of Illness Index; Sodium Channels; Substance Withdrawal Syndrome | 2007 |