phenylalanine has been researched along with Bone Marrow Neoplasms in 6 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 6 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Dirnhofer, S; Duek, A; Grisouard, J; Hao-Shen, H; Kubovcakova, L; Shimizu, T; Skoda, RC | 1 |
| Kirito, K; Koda, AH; Komatsu, N; Morishita, S; Noda, N; Sekiguchi, Y; Tsuneda, S | 1 |
| Barosi, G; Bergamaschi, G; Carolei, A; Catarsi, P; Poletto, V; Primignani, M; Rosti, V; Spolverini, A; Vannucchi, AM; Villani, L | 1 |
| Iványi, JL; Marton, E; Plander, M | 1 |
| Bisht, KS; Cogle, CR; Gali, M; Keserü, GM; Kirabo, A; Majumder, A; Park, SO; Reinhard, MK; Sayeski, PP; Wamsley, HL; Zhao, ZJ | 1 |
| Ahmed, W; Betancur, M; Hennighausen, L; Lazarides, K; Patel, N; Van Etten, RA; Walz, C; Zaleskas, VM | 1 |
6 other study(ies) available for phenylalanine and Bone Marrow Neoplasms
| Article | Year |
|---|---|
Deletion of Stat3 in hematopoietic cells enhances thrombocytosis and shortens survival in a JAK2-V617F mouse model of MPN.
Topics: Amino Acid Substitution; Animals; Bone Marrow; Bone Marrow Neoplasms; Disease Models, Animal; Disease Progression; Gene Deletion; Hematopoietic Stem Cells; Janus Kinase 2; Mice; Mice, Inbred C57BL; Mice, Transgenic; Myeloproliferative Disorders; Phenylalanine; STAT3 Transcription Factor; Thrombocytosis; Valine | 2015 |
Alternately binding probe competitive PCR as a simple, cost-effective, and accurate quantification method for JAK2V617F allele burden in myeloproliferative neoplasms.
Topics: Alleles; Amino Acid Substitution; Binding, Competitive; Bone Marrow Neoplasms; Cell Line, Tumor; Cost-Benefit Analysis; DNA Mutational Analysis; DNA Probes; Gene Dosage; Humans; Janus Kinase 2; Models, Biological; Myeloproliferative Disorders; Phenylalanine; Polymerase Chain Reaction; Reference Standards; Reproducibility of Results; Sensitivity and Specificity; Valine | 2011 |
JAK2 46/1 haplotype predisposes to splanchnic vein thrombosis-associated BCR-ABL negative classic myeloproliferative neoplasms.
Topics: Bone Marrow Neoplasms; Case-Control Studies; Fusion Proteins, bcr-abl; Gene Frequency; Genetic Predisposition to Disease; Haplotypes; Humans; Janus Kinase 2; Mesenteric Vascular Occlusion; Mesenteric Veins; Mutation; Myeloproliferative Disorders; Phenylalanine; Polymorphism, Genetic; Polymorphism, Single Nucleotide; Splanchnic Circulation; Valine; Venous Thrombosis | 2012 |
[Significance of the JAK2V617F mutation in patients with chronic myeloproliferative neoplasia].
Topics: Adult; Aged; Bone Marrow Neoplasms; Chronic Disease; Female; Humans; Janus Kinase 2; Male; Middle Aged; Mutation; Myeloproliferative Disorders; Phenylalanine; Polycythemia Vera; Polymerase Chain Reaction; Predictive Value of Tests; Primary Myelofibrosis; Retrospective Studies; Signal Transduction; Thrombocythemia, Essential; Thrombosis; Valine | 2011 |
The Jak2 inhibitor, G6, alleviates Jak2-V617F-mediated myeloproliferative neoplasia by providing significant therapeutic efficacy to the bone marrow.
Topics: Amino Acid Substitution; Animals; Antineoplastic Agents; Bone Marrow Cells; Bone Marrow Neoplasms; Cell Transformation, Neoplastic; Cells, Cultured; Drug Evaluation, Preclinical; Janus Kinase 2; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Myeloproliferative Disorders; Phenylalanine; Protein Kinase Inhibitors; Stilbenes; Valine | 2011 |
Essential role for Stat5a/b in myeloproliferative neoplasms induced by BCR-ABL1 and JAK2(V617F) in mice.
Topics: Amino Acid Substitution; Animals; Bone Marrow Neoplasms; Cell Transformation, Neoplastic; Genes, abl; HEK293 Cells; Humans; Janus Kinase 2; Mice; Mice, Congenic; Mice, Inbred BALB C; Mice, Transgenic; Mutation, Missense; Myeloproliferative Disorders; NIH 3T3 Cells; Phenylalanine; STAT5 Transcription Factor; Valine | 2012 |