phenobarbital has been researched along with Cancer, Radiation-Induced in 7 studies
Phenobarbital: A barbituric acid derivative that acts as a nonselective central nervous system depressant. It potentiates GAMMA-AMINOBUTYRIC ACID action on GABA-A RECEPTORS, and modulates chloride currents through receptor channels. It also inhibits glutamate induced depolarizations.
phenobarbital : A member of the class of barbiturates, the structure of which is that of barbituric acid substituted at C-5 by ethyl and phenyl groups.
| Excerpt | Relevance | Reference |
|---|---|---|
| " The public health implication of this lack of linearity in the low-dose area of the dose-response curve raises the question of whether low doses of carcinogens will reduce cancer risk." | 1.30 | Can the concept of hormesis Be generalized to carcinogenesis? ( Baldwin, LA; Calabrese, EJ, 1998) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 4 (57.14) | 18.7374 |
| 1990's | 2 (28.57) | 18.2507 |
| 2000's | 1 (14.29) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Kapadia, GJ | 1 |
| Azuine, MA | 1 |
| Sridhar, R | 1 |
| Okuda, Y | 1 |
| Tsuruta, A | 1 |
| Ichiishi, E | 1 |
| Mukainake, T | 1 |
| Takasaki, M | 1 |
| Konoshima, T | 1 |
| Nishino, H | 1 |
| Tokuda, H | 1 |
| Kim, SH | 1 |
| Lee, YS | 1 |
| Lee, MS | 1 |
| Kim, TH | 1 |
| Jang, JJ | 1 |
| Calabrese, EJ | 1 |
| Baldwin, LA | 1 |
| Black, HS | 1 |
| Chan, JT | 1 |
| Brown, GE | 1 |
| Kaufmann, WK | 2 |
| MacKenzie, SA | 2 |
| Kaufman, DG | 2 |
| Rahija, RJ | 1 |
| Trosko, JE | 1 |
| Chang, CC | 1 |
7 other studies available for phenobarbital and Cancer, Radiation-Induced
| Article | Year |
|---|---|
Chemoprevention of DMBA-induced UV-B promoted, NOR-1-induced TPA promoted skin carcinogenesis, and DEN-induced phenobarbital promoted liver tumors in mice by extract of beetroot.
Topics: 9,10-Dimethyl-1,2-benzanthracene; Animals; Anticarcinogenic Agents; Beta vulgaris; Betacyanins; Body | 2003 |
Development of altered hepatocyte foci by separate and combined treatments with radiation and diethylnitrosamine in neonatal rats.
Topics: Animals; Body Weight; Diethylnitrosamine; Female; Gamma Rays; Glutathione Transferase; Liver; Liver | 1994 |
Can the concept of hormesis Be generalized to carcinogenesis?
Topics: Animals; Caffeic Acids; Carcinogenicity Tests; Carcinogens; Cell Division; Dioxins; DNA Ligases; Dos | 1998 |
Effects of dietary constituents on ultraviolet light-mediated carcinogenesis.
Topics: Animals; Antioxidants; Butylated Hydroxytoluene; Diet; Disulfiram; Female; Glutathione; Keratosis; M | 1978 |
Factors influencing the initiation by gamma rays of hepatocarcinogenesis in the rat.
Topics: Animals; Carcinoma, Hepatocellular; Cell Cycle; Cells, Cultured; DNA Repair; Dose-Response Relations | 1987 |
Quantitative relationship between initiation of hepatocarcinogenesis and induction of altered cell islands.
Topics: 7,8-Dihydro-7,8-dihydroxybenzo(a)pyrene 9,10-oxide; Animals; Benzopyrenes; Dimethylnitrosamine; DNA; | 1986 |
Role of tumor promotion in affecting the multi-hit nature of carcinogenesis.
Topics: Animals; Carcinogens; Carcinoma, Squamous Cell; Cell Division; Cell Transformation, Neoplastic; Fema | 1985 |