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phenethyl isothiocyanate and Mouth Neoplasms

phenethyl isothiocyanate has been researched along with Mouth Neoplasms in 9 studies

Research

Studies (9)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (11.11)18.2507
2000's2 (22.22)29.6817
2010's5 (55.56)24.3611
2020's1 (11.11)2.80

Authors

AuthorsStudies
Krishnan, M; Venkidasamy, B1
Fitzgerald, AL; Huang, P; Lam-Ubol, A; Myers, JN; Phonyiam, T; Ritdej, A; Trachootham, D; Zhang, H1
Amagaya, S; Chen, HJ; Lee, CY; Lin, CM; Lin, YC; Shih, NC; Yang, JS1
Chen, ZF; Huang, SY; Lee, KJ; Lin, JS; Shyu, HW; Su, SH; Su, SJ; Yeh, H; Yeh, YT1
Deng, YT; Hsieh, YP; Ko, HH; Kuo, MY; Wu, KJ; Yeh, CC1
Cho, NP; Cho, SD; Choi, ES; Huong, LD; Kim, HM; Leem, DH; Shin, JA1
Chang, Kw; Helenowski, I; Rademaker, AW; Solt, DB1
Chang, KW; Chiu, PC; Raghian, A; Shabany, K; Solt, DB1
Bowman, D; Davis, WJ; Heiblum, R; King, PG; Murphy, SE; Stoner, GD1

Other Studies

9 other study(ies) available for phenethyl isothiocyanate and Mouth Neoplasms

ArticleYear
PEITC: A prospective natural metabolite in oral cancer treatment.
    Oral oncology, 2022, Volume: 133

    Topics: Anticarcinogenic Agents; Apoptosis; Cell Line, Tumor; Humans; Isothiocyanates; Mouth Neoplasms; Prospective Studies

2022
Sensory acceptable equivalent doses of β-phenylethyl isothiocyanate (PEITC) induce cell cycle arrest and retard the growth of p53 mutated oral cancer in vitro and in vivo.
    Food & function, 2018, Jul-17, Volume: 9, Issue:7

    Topics: Animals; Anticarcinogenic Agents; Apoptosis; Cell Cycle Checkpoints; Cell Line, Tumor; Cell Proliferation; Humans; Isothiocyanates; Male; Mice; Mice, Nude; Mouth Neoplasms; Taste; Tumor Suppressor Protein p53

2018
Phenethyl isothiocyanate suppresses EGF-stimulated SAS human oral squamous carcinoma cell invasion by targeting EGF receptor signaling.
    International journal of oncology, 2013, Volume: 43, Issue:2

    Topics: Anticarcinogenic Agents; Carcinoma, Squamous Cell; Cell Line, Tumor; Cell Survival; Epidermal Growth Factor; ErbB Receptors; Humans; I-kappa B Kinase; Isothiocyanates; MAP Kinase Signaling System; Matrix Metalloproteinase 2; Matrix Metalloproteinase 9; Mouth Neoplasms; Neoplasm Invasiveness; NF-kappa B; Phosphatidylinositol 3-Kinases; Phosphorylation; Protein Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt; Pyruvate Dehydrogenase Acetyl-Transferring Kinase; Signal Transduction; Tissue Inhibitor of Metalloproteinase-1; Tissue Inhibitor of Metalloproteinase-2

2013
Phenethyl isothiocyanate induces DNA damage-associated G2/M arrest and subsequent apoptosis in oral cancer cells with varying p53 mutations.
    Free radical biology & medicine, 2014, Volume: 74

    Topics: Anticarcinogenic Agents; Apoptosis; Ataxia Telangiectasia Mutated Proteins; Brassicaceae; Carcinoma, Squamous Cell; Cell Cycle Proteins; Cell Line, Tumor; Checkpoint Kinase 2; DNA Damage; G2 Phase Cell Cycle Checkpoints; Gene Expression Regulation, Neoplastic; Glutathione; Growth Inhibitors; Humans; Isothiocyanates; Membrane Potentials; Mitochondria; Mouth Neoplasms; Mutation; Nitric Oxide; Oxidative Stress; Reactive Oxygen Species; Tumor Suppressor Protein p53

2014
Phenethyl isothiocyanate enhances TRAIL-induced apoptosis in oral cancer cells and xenografts.
    Clinical oral investigations, 2016, Volume: 20, Issue:9

    Topics: Acetylcysteine; Animals; Anthracenes; Apoptosis; Blotting, Western; Cell Line, Tumor; Cell Survival; Drug Resistance, Neoplasm; Heterografts; Humans; In Situ Nick-End Labeling; Isothiocyanates; Mice; Mouth Neoplasms; Reactive Oxygen Species; Receptors, TNF-Related Apoptosis-Inducing Ligand; TNF-Related Apoptosis-Inducing Ligand

2016
β-Phenethyl isothiocyanate induces death receptor 5 to induce apoptosis in human oral cancer cells via p38.
    Oral diseases, 2012, Volume: 18, Issue:5

    Topics: Apoptosis; Caspase 3; Caspase 8; Cell Line, Tumor; Cell Proliferation; Enzyme Inhibitors; Humans; Isothiocyanates; MAP Kinase Signaling System; Mouth Neoplasms; p38 Mitogen-Activated Protein Kinases; Receptors, TNF-Related Apoptosis-Inducing Ligand

2012
Phenethyl isothiocyanate inhibits nitrosamine carcinogenesis in a model for study of oral cancer chemoprevention.
    Cancer letters, 2003, Dec-30, Volume: 202, Issue:2

    Topics: Animals; Anticarcinogenic Agents; Biomarkers, Tumor; Cricetinae; Disease Models, Animal; DNA Replication; Dose-Response Relationship, Drug; gamma-Glutamyltransferase; Isothiocyanates; Male; Mesocricetus; Mouth Mucosa; Mouth Neoplasms; Nitrosamines

2003
Rapid in vivo assay for topical oral cancer chemopreventive agents.
    International journal of oncology, 2002, Volume: 21, Issue:1

    Topics: Administration, Oral; Aldehyde Dehydrogenase; Animals; Biological Assay; Carcinogens; Carcinoma, Squamous Cell; Corn Oil; Cricetinae; Dimethylnitrosamine; gamma-Glutamyltransferase; Genes, p53; Isothiocyanates; Male; Mesocricetus; Mouth Mucosa; Mouth Neoplasms

2002
Effect of phenethyl isothiocyanate on the metabolism of tobacco-specific nitrosamines by cultured rat oral tissue.
    Carcinogenesis, 1991, Volume: 12, Issue:6

    Topics: Animals; Carcinogens; Culture Techniques; Hydroxylation; Isothiocyanates; Male; Mouth Mucosa; Mouth Neoplasms; Nitrosamines; Plants, Toxic; Rats; Rats, Inbred F344; Thiocyanates; Tobacco, Smokeless

1991