peptide elongation factor 2 has been researched along with Cell Transformation, Neoplastic in 5 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (20.00) | 18.7374 |
| 1990's | 2 (40.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (40.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Agnihotri, S; Ahern, CA; Barokas, A; Bowden, M; Bridon, G; Collins, VP; Delattre, O; Derry, WB; Dubuc, A; El-Naggar, A; Faubert, B; Galpin, JD; Gleave, ME; Hielscher, T; Jan, E; Jones, RG; Kool, M; Korshunov, A; Kwok, B; Leprivier, G; Li, A; Mateo, AR; Mathers, J; McCabe, MG; Northcott, PA; Pfister, SM; Pollak, M; Proud, CG; Remke, M; Rotblat, B; Smith, S; Somasekharan, SP; Sorensen, PH; Taylor, MD; Thomas, R; Tognon, CE; Wang, Y; Wu, X; Yu, B | 1 |
| Bushell, M; Casey, HA; Cordero, JB; Dudek, KM; Faller, WJ; Hall, MN; Huels, DJ; Jackson, TJ; Jamieson, T; Jones, C; Karim, SA; Knight, JR; Meyuhas, O; Myant, KB; Pende, M; Radulescu, S; Ridgway, RA; Ryazanov, AG; Sansom, OJ; Scopelliti, A; Sonenberg, N; Vidal, M; Willis, AE | 1 |
| Almasan, A; Huang, LC; Linke, SP; Paulson, TG; Wahl, GM | 1 |
| Alaiya, AA; Appella, E; Auer, G; Bergman, AC; Bergman, T; Binetruy, B; Jörnvall, H; Kronenwett, U; Linder, S; Sakaguchi, K; Shoshan, M; Wendler, W | 1 |
| Gschwendt, M; Kittstein, W; Marks, F | 1 |
1 review(s) available for peptide elongation factor 2 and Cell Transformation, Neoplastic
| Article | Year |
|---|---|
Genetic instability as a consequence of inappropriate entry into and progression through S-phase.
Topics: Animals; Antimetabolites; Apoptosis; Cell Transformation, Neoplastic; Chromosome Aberrations; Cyclin-Dependent Kinase Inhibitor p21; Cyclins; DNA Damage; G1 Phase; Gene Expression Regulation; Genes, p53; Genes, Retinoblastoma; Genome; Humans; Mutation; Neoplasms; Peptide Elongation Factor 2; Peptide Elongation Factors; Protein Kinase Inhibitors; Protein Kinases; Retinoblastoma Protein; S Phase; Tumor Suppressor Protein p53 | 1995 |
4 other study(ies) available for peptide elongation factor 2 and Cell Transformation, Neoplastic
| Article | Year |
|---|---|
The eEF2 kinase confers resistance to nutrient deprivation by blocking translation elongation.
Topics: AMP-Activated Protein Kinases; Animals; Brain Neoplasms; Caenorhabditis elegans; Cell Survival; Cell Transformation, Neoplastic; Elongation Factor 2 Kinase; Food Deprivation; Glioblastoma; HeLa Cells; Humans; Mice; Mice, Nude; Neoplasm Transplantation; Neoplasms; NIH 3T3 Cells; Peptide Chain Elongation, Translational; Peptide Elongation Factor 2; Signal Transduction; Transplantation, Heterologous | 2013 |
mTORC1-mediated translational elongation limits intestinal tumour initiation and growth.
Topics: Adenomatous Polyposis Coli Protein; Animals; Cell Proliferation; Cell Transformation, Neoplastic; Elongation Factor 2 Kinase; Enzyme Activation; Genes, APC; Intestinal Neoplasms; Male; Mechanistic Target of Rapamycin Complex 1; Mice; Mice, Inbred C57BL; Multiprotein Complexes; Oncogene Protein p55(v-myc); Peptide Chain Elongation, Translational; Peptide Elongation Factor 2; Ribosomal Protein S6 Kinases; Signal Transduction; TOR Serine-Threonine Kinases; Wnt Proteins | 2015 |
Protein kinase-dependent overexpression of the nuclear protein pirin in c-JUN and RAS transformed fibroblasts.
Topics: Animals; Calcium-Calmodulin-Dependent Protein Kinases; Carrier Proteins; Cell Line, Transformed; Cell Transformation, Neoplastic; Dioxygenases; Fibroblasts; Flavonoids; Genes, jun; Genes, ras; Heterogeneous-Nuclear Ribonucleoproteins; MAP Kinase Kinase 1; Mass Spectrometry; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinase Kinases; Mitogen-Activated Protein Kinases; Nuclear Proteins; Ornithine-Oxo-Acid Transaminase; Peptide Elongation Factor 2; Peptide Elongation Factors; Phosphoglycerate Kinase; Phosphopyruvate Hydratase; Protein Serine-Threonine Kinases; Protein-Tyrosine Kinases; Proto-Oncogene Proteins c-raf; Rats; Ribonucleoproteins; Signal Transduction; Transfection | 1999 |
Ciclosporin inhibits phorbol-ester-induced hyperplastic transformation and tumor promotion in mouse skin probably by suppression of Ca2+/calmodulin-dependent processes such as phosphorylation of elongation factor 2.
Topics: Animals; Calcium; Calmodulin; Cell Transformation, Neoplastic; Chromatography, DEAE-Cellulose; Cocarcinogenesis; Cycloheximide; Cyclosporins; Cytosol; Electrophoresis, Polyacrylamide Gel; Mice; Peptide Elongation Factor 2; Peptide Elongation Factors; Phosphorus Radioisotopes; Phosphorylation; Skin; Skin Neoplasms; Tetradecanoylphorbol Acetate | 1988 |