Page last updated: 2024-11-07

penicillamine and Leukemia, Myelogenous, Chronic, BCR-ABL Positive

penicillamine has been researched along with Leukemia, Myelogenous, Chronic, BCR-ABL Positive in 2 studies

Penicillamine: 3-Mercapto-D-valine. The most characteristic degradation product of the penicillin antibiotics. It is used as an antirheumatic and as a chelating agent in Wilson's disease.
penicillamine : An alpha-amino acid having the structure of valine substituted at the beta position with a sulfanyl group.

Leukemia, Myelogenous, Chronic, BCR-ABL Positive: Clonal hematopoetic disorder caused by an acquired genetic defect in PLURIPOTENT STEM CELLS. It starts in MYELOID CELLS of the bone marrow, invades the blood and then other organs. The condition progresses from a stable, more indolent, chronic phase (LEUKEMIA, MYELOID, CHRONIC PHASE) lasting up to 7 years, to an advanced phase composed of an accelerated phase (LEUKEMIA, MYELOID, ACCELERATED PHASE) and BLAST CRISIS.

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Kaşifoğlu, T1
Korkmaz, C1
Yaşar, S1
Gülbaş, Z1
Ferry-Dumazet, H1
Mamani-Matsuda, M1
Dupouy, M1
Belloc, F1
Thiolat, D1
Marit, G1
Arock, M1
Reiffers, J1
Mossalayi, MD1

Other Studies

2 other studies available for penicillamine and Leukemia, Myelogenous, Chronic, BCR-ABL Positive

ArticleYear
Scleroderma and chronic myeloid leukemia: a sheer coincidence, a consequence of long lasting D-penicillamine therapy or a plausible relationship of both diseases?
    Rheumatology international, 2006, Volume: 27, Issue:2

    Topics: Antirheumatic Agents; CREST Syndrome; Female; Humans; Leukemia, Myelogenous, Chronic, BCR-ABL Positi

2006
Nitric oxide induces the apoptosis of human BCR-ABL-positive myeloid leukemia cells: evidence for the chelation of intracellular iron.
    Leukemia, 2002, Volume: 16, Issue:4

    Topics: Apoptosis; Cell Cycle; Cell Division; Cells, Cultured; Ferricyanides; Fusion Proteins, bcr-abl; Huma

2002