Page last updated: 2024-11-02

pd 98059 and Carbon Tetrachloride Poisoning

pd 98059 has been researched along with Carbon Tetrachloride Poisoning in 2 studies

2-(2-amino-3-methoxyphenyl)-4H-1-benzopyran-4-one: inhibits MAP kinase kinase (MEK) activity, p42 MAPK and p44 MAPK; structure in first source
2-(2-amino-3-methoxyphenyl)chromen-4-one : A member of the class of monomethoxyflavones that is 3'-methoxyflavone bearing an additional amino substituent at position 2'.

Carbon Tetrachloride Poisoning: Poisoning that results from ingestion, injection, inhalation, or skin absorption of CARBON TETRACHLORIDE.

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Smart, DE1
Green, K1
Oakley, F1
Weitzman, JB1
Yaniv, M1
Reynolds, G1
Mann, J1
Millward-Sadler, H1
Mann, DA1
Kawao, N1
Okada, K1
Kawata, S1
Okamoto, C1
Tsuritani, M1
Ueshima, S1
Matsuo, O1

Other Studies

2 other studies available for pd 98059 and Carbon Tetrachloride Poisoning

ArticleYear
JunD is a profibrogenic transcription factor regulated by Jun N-terminal kinase-independent phosphorylation.
    Hepatology (Baltimore, Md.), 2006, Volume: 44, Issue:6

    Topics: Actins; Alanine Transaminase; Animals; Anthracenes; Carbon Tetrachloride Poisoning; Extracellular Si

2006
Plasmin decreases the BH3-only protein BimEL via the ERK1/2 signaling pathway in hepatocytes.
    Biochimica et biophysica acta, 2007, Volume: 1773, Issue:6

    Topics: Aminocaproic Acid; Animals; Antifibrinolytic Agents; Apoptosis Regulatory Proteins; Aprotinin; Bcl-2

2007