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pd 98059 and Cancer of Nasopharynx

pd 98059 has been researched along with Cancer of Nasopharynx in 5 studies

2-(2-amino-3-methoxyphenyl)-4H-1-benzopyran-4-one: inhibits MAP kinase kinase (MEK) activity, p42 MAPK and p44 MAPK; structure in first source
2-(2-amino-3-methoxyphenyl)chromen-4-one : A member of the class of monomethoxyflavones that is 3'-methoxyflavone bearing an additional amino substituent at position 2'.

Research Excerpts

ExcerptRelevanceReference
"Curcumin, one of the main bioactive components extracted from a traditional Chinese medicinal herb, exhibits potent anticancer activity against many types of cancer cells including nasopharyngeal carcinoma (NPC)."7.80Extracellular signal-regulated kinase signaling-mediated induction and interaction of FOXO3a and p53 contribute to the inhibition of nasopharyngeal carcinoma cell growth by curcumin. ( Hahn, SS; Tang, G; Tang, Q; Wu, J; Wu, Y; Zhao, S; Zheng, F, 2014)
" Wogonin, a flavonoid compound derived from the traditional Chinese medicine of Huang-Qin, has anticancer activity in many cancer cells including human nasopharyngeal carcinoma (NPC)."7.78Wogonin induces cross-regulation between autophagy and apoptosis via a variety of Akt pathway in human nasopharyngeal carcinoma cells. ( Chen, YW; Chow, SE; Huang, YK; Liang, CA; Wang, JS, 2012)
"Curcumin, one of the main bioactive components extracted from a traditional Chinese medicinal herb, exhibits potent anticancer activity against many types of cancer cells including nasopharyngeal carcinoma (NPC)."3.80Extracellular signal-regulated kinase signaling-mediated induction and interaction of FOXO3a and p53 contribute to the inhibition of nasopharyngeal carcinoma cell growth by curcumin. ( Hahn, SS; Tang, G; Tang, Q; Wu, J; Wu, Y; Zhao, S; Zheng, F, 2014)
" Wogonin, a flavonoid compound derived from the traditional Chinese medicine of Huang-Qin, has anticancer activity in many cancer cells including human nasopharyngeal carcinoma (NPC)."3.78Wogonin induces cross-regulation between autophagy and apoptosis via a variety of Akt pathway in human nasopharyngeal carcinoma cells. ( Chen, YW; Chow, SE; Huang, YK; Liang, CA; Wang, JS, 2012)

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (20.00)29.6817
2010's4 (80.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Wu, J1
Tang, Q1
Zhao, S1
Zheng, F1
Wu, Y1
Tang, G1
Hahn, SS1
Chen, LC1
Liu, HP1
Li, HP1
Hsueh, C1
Yu, JS1
Liang, CL1
Chang, YS1
Liu, H2
Duan, Z1
Zheng, H1
Hu, D1
Li, M1
Tao, Y2
Bode, AM2
Dong, Z2
Cao, Y2
Chow, SE1
Chen, YW1
Liang, CA1
Huang, YK1
Wang, JS1
Liu, S1
Li, H1
Chen, L1
Yang, L1
Li, L1
Li, W1
Li, Z1
Tang, M1

Other Studies

5 other studies available for pd 98059 and Cancer of Nasopharynx

ArticleYear
Extracellular signal-regulated kinase signaling-mediated induction and interaction of FOXO3a and p53 contribute to the inhibition of nasopharyngeal carcinoma cell growth by curcumin.
    International journal of oncology, 2014, Volume: 45, Issue:1

    Topics: Antineoplastic Agents; Apoptosis; Carcinoma; Cell Line, Tumor; Cell Proliferation; Curcumin; Dose-Re

2014
Thymidine phosphorylase mRNA stability and protein levels are increased through ERK-mediated cytoplasmic accumulation of hnRNP K in nasopharyngeal carcinoma cells.
    Oncogene, 2009, Apr-30, Volume: 28, Issue:17

    Topics: Base Sequence; Blotting, Western; Cell Hypoxia; Cell Line, Tumor; Culture Media, Serum-Free; Cytopla

2009
EBV-encoded LMP1 upregulates Igκ 3'enhancer activity and Igκ expression in nasopharyngeal cancer cells by activating the Ets-1 through ERKs signaling.
    PloS one, 2012, Volume: 7, Issue:3

    Topics: Binding Sites; Carcinoma; Cell Line, Tumor; DNA Mutational Analysis; Enzyme Inhibitors; Extracellula

2012
Wogonin induces cross-regulation between autophagy and apoptosis via a variety of Akt pathway in human nasopharyngeal carcinoma cells.
    Journal of cellular biochemistry, 2012, Volume: 113, Issue:11

    Topics: Adenine; Apoptosis; Autophagy; Carcinoma; Cell Line, Tumor; Drugs, Chinese Herbal; Extracellular Sig

2012
(-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous lytic infection involves ERK1/2 and PI3-K/Akt signaling in EBV-positive cells.
    Carcinogenesis, 2013, Volume: 34, Issue:3

    Topics: Antiviral Agents; Catechin; Cell Line, Tumor; Cell Survival; Chromones; DNA, Viral; Enzyme Activatio

2013