Page last updated: 2024-10-21

pd 173074 and Uremia

pd 173074 has been researched along with Uremia in 2 studies

Uremia: A clinical syndrome associated with the retention of renal waste products or uremic toxins in the blood. It is usually the result of RENAL INSUFFICIENCY. Most uremic toxins are end products of protein or nitrogen CATABOLISM, such as UREA or CREATININE. Severe uremia can lead to multiple organ dysfunctions with a constellation of symptoms.

Research Excerpts

ExcerptRelevanceReference
" Folic acid-induced acute kidney injury increased calvaria FGF23 mRNA and serum FGF23 and parathyroid hormone (PTH) levels at 6 h."3.83The fibroblast growth factor receptor mediates the increased FGF23 expression in acute and chronic uremia. ( Durlacher, K; Hassan, A; Levi, R; Naveh-Many, T; Silver, J, 2016)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Czaya, B1
Seeherunvong, W1
Singh, S1
Yanucil, C1
Ruiz, P1
Quiroz, Y1
Grabner, A1
Katsoufis, C1
Swaminathan, S1
Abitbol, C1
Rodriguez-Iturbe, B1
Faul, C1
Freundlich, M1
Hassan, A1
Durlacher, K1
Silver, J1
Naveh-Many, T1
Levi, R1

Other Studies

2 other studies available for pd 173074 and Uremia

ArticleYear
Cardioprotective Effects of Paricalcitol Alone and in Combination With FGF23 Receptor Inhibition in Chronic Renal Failure: Experimental and Clinical Studies.
    American journal of hypertension, 2019, 01-01, Volume: 32, Issue:1

    Topics: Adolescent; Animals; Cardio-Renal Syndrome; Cardiomyopathies; Child; Disease Models, Animal; Drug Th

2019
The fibroblast growth factor receptor mediates the increased FGF23 expression in acute and chronic uremia.
    American journal of physiology. Renal physiology, 2016, Feb-01, Volume: 310, Issue:3

    Topics: Acute Kidney Injury; Adenine; Animals; Chronic Disease; Disease Models, Animal; Fibroblast Growth Fa

2016
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