Page last updated: 2024-08-26

paxilline and Nephrotic Syndrome

paxilline has been researched along with Nephrotic Syndrome in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	1 (50.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Chesor, M; Lait, PJ; Lee, RWJ; May, CJ; Saleem, MA; Schewitz-Bowers, LP; Welsh, GI	1
Koukouritaki, SB; Lianos, EA; Tamizuddin, A	1

Other Studies

2 other study(ies) available for paxilline and Nephrotic Syndrome

Article	Year
Human Th17 cells produce a soluble mediator that increases podocyte motility via signaling pathways that mimic PAR-1 activation. American journal of physiology. Renal physiology, 2019, 10-01, Volume: 317, Issue:4 Topics: Cell Movement; Cells, Cultured; Healthy Volunteers; Humans; Interleukin-17; Nephrotic Syndrome; p38 Mitogen-Activated Protein Kinases; Paxillin; Podocytes; Protease Inhibitors; Protein Serine-Threonine Kinases; Signal Transduction; Th17 Cells	2019
Enhanced expression of the cytoskeletal-associated protein, paxillin, in experimental nephrotic syndrome. Journal of investigative medicine : the official publication of the American Federation for Clinical Research, 1998, Volume: 46, Issue:6 Topics: Animals; Anti-Bacterial Agents; Blotting, Western; Cell Adhesion Molecules; Cells, Cultured; Cytoskeletal Proteins; Disease Models, Animal; Focal Adhesion Kinase 1; Focal Adhesion Protein-Tyrosine Kinases; Follow-Up Studies; Nephrotic Syndrome; Paxillin; Phosphoproteins; Protein-Tyrosine Kinases; Puromycin; Rats; Rats, Sprague-Dawley; Receptor, Insulin; rho GTP-Binding Proteins	1998

Article

Year

Human Th17 cells produce a soluble mediator that increases podocyte motility via signaling pathways that mimic PAR-1 activation.

American journal of physiology. Renal physiology, 2019, 10-01, Volume: 317, Issue:4

Topics: Cell Movement; Cells, Cultured; Healthy Volunteers; Humans; Interleukin-17; Nephrotic Syndrome; p38 Mitogen-Activated Protein Kinases; Paxillin; Podocytes; Protease Inhibitors; Protein Serine-Threonine Kinases; Signal Transduction; Th17 Cells

2019

Enhanced expression of the cytoskeletal-associated protein, paxillin, in experimental nephrotic syndrome.

Journal of investigative medicine : the official publication of the American Federation for Clinical Research, 1998, Volume: 46, Issue:6

Topics: Animals; Anti-Bacterial Agents; Blotting, Western; Cell Adhesion Molecules; Cells, Cultured; Cytoskeletal Proteins; Disease Models, Animal; Focal Adhesion Kinase 1; Focal Adhesion Protein-Tyrosine Kinases; Follow-Up Studies; Nephrotic Syndrome; Paxillin; Phosphoproteins; Protein-Tyrosine Kinases; Puromycin; Rats; Rats, Sprague-Dawley; Receptor, Insulin; rho GTP-Binding Proteins

1998