paroxetine has been researched along with Ventricular Dysfunction, Left in 1 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bhuiyan, S; Fukunaga, K; Hasegawa, H; Kanai, H; Shioda, N; Tagashira, H | 1 |
1 other study(ies) available for paroxetine and Ventricular Dysfunction, Left
| Article | Year |
|---|---|
Sigma1-receptor stimulation with fluvoxamine ameliorates transverse aortic constriction-induced myocardial hypertrophy and dysfunction in mice.
Topics: Animals; Aorta; Cells, Cultured; Fluvoxamine; Hypertrophy, Left Ventricular; Male; Mice; Mice, Inbred ICR; Models, Animal; Myocardial Contraction; Myocytes, Cardiac; Nitric Oxide Synthase Type III; Paroxetine; Phosphorylation; Proto-Oncogene Proteins c-akt; Rats; Rats, Wistar; Receptors, sigma; Selective Serotonin Reuptake Inhibitors; Sigma-1 Receptor; Time Factors; Vasoconstriction; Ventricular Dysfunction, Left | 2010 |