paraquat has been researched along with Ventricular Dysfunction, Left in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Feng, Q; Wang, T; Zhang, L | 1 |
| Hua, Y; Nair, S; Ren, J; Wang, Q; Xu, X; Yang, L | 1 |
2 other study(ies) available for paraquat and Ventricular Dysfunction, Left
| Article | Year |
|---|---|
Necrostatin-1 Protects Against Paraquat-Induced Cardiac Contractile Dysfunction via RIP1-RIP3-MLKL-Dependent Necroptosis Pathway.
Topics: Animals; Antioxidants; Calcium Signaling; Cardiotoxicity; Cell Death; Cytoprotection; Disease Models, Animal; GTPase-Activating Proteins; Imidazoles; Indoles; Mice, Inbred C57BL; Myocardial Contraction; Myocytes, Cardiac; Oxidative Stress; Paraquat; Protein Kinases; Reactive Oxygen Species; Receptor-Interacting Protein Serine-Threonine Kinases; Ventricular Dysfunction, Left; Ventricular Function, Left | 2018 |
AMP-activated protein kinase deficiency rescues paraquat-induced cardiac contractile dysfunction through an autophagy-dependent mechanism.
Topics: AMP-Activated Protein Kinases; Animals; Autophagy; Calcium; Echocardiography; Female; Herbicides; Male; Membrane Potential, Mitochondrial; Mice, Inbred C57BL; Mice, Transgenic; Myocardial Contraction; Myocytes, Cardiac; Paraquat; Ventricular Dysfunction, Left | 2014 |