Page last updated: 2024-08-21

paraquat and Cardiac Toxicity

paraquat has been researched along with Cardiac Toxicity in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's5 (83.33)24.3611
2020's1 (16.67)2.80

Authors

AuthorsStudies
Alenabi, A; Hallaj-Salahipour, M; Khoramjouy, M; Malekinejad, H; Malekinejad, M; Masoumi Verki, M1
Chang, GJ; Hsu, KH; Lin, CC; Shih, CP1
Feng, Q; Wang, T; Zhang, L1
Chen, WL; Peng, CG; Wang, DW; Xiao, XY; Xie, S; Zhang, WX1
Gao, B; Hu, N; Li, N; Liu, M; Lu, S; Wang, H; Wang, J; Yu, W; Zhang, G; Zhang, Y; Zheng, Q1
Ren, J; Wang, S; Xiong, L; Zhang, Y; Zhu, X1

Other Studies

6 other study(ies) available for paraquat and Cardiac Toxicity

ArticleYear
Cardioprotective Effects of Atorvastatin Are Mediated Through PPARĪ³ in Paraquat-Exposed Rats.
    Journal of cardiovascular pharmacology, 2019, Volume: 74, Issue:5

    Topics: Animals; Antioxidants; Atorvastatin; Cardiotoxicity; Creatine Kinase, MB Form; Cyclooxygenase 2; Disease Models, Animal; Heart Diseases; Lipid Peroxidation; Male; Myocytes, Cardiac; Nitric Oxide; Oxidative Stress; Paraquat; Peroxidase; PPAR gamma; Rats, Wistar; Signal Transduction; Sulfhydryl Compounds

2019
Hemodynamic and electromechanical effects of paraquat in rat heart.
    PloS one, 2021, Volume: 16, Issue:4

    Topics: Action Potentials; Animals; Cardiotoxicity; Cells, Cultured; Heart; Heart Rate; Hemodynamics; Herbicides; Male; Models, Animal; Myocardial Contraction; Myocytes, Cardiac; Paraquat; Rats; Rats, Sprague-Dawley

2021
Necrostatin-1 Protects Against Paraquat-Induced Cardiac Contractile Dysfunction via RIP1-RIP3-MLKL-Dependent Necroptosis Pathway.
    Cardiovascular toxicology, 2018, Volume: 18, Issue:4

    Topics: Animals; Antioxidants; Calcium Signaling; Cardiotoxicity; Cell Death; Cytoprotection; Disease Models, Animal; GTPase-Activating Proteins; Imidazoles; Indoles; Mice, Inbred C57BL; Myocardial Contraction; Myocytes, Cardiac; Oxidative Stress; Paraquat; Protein Kinases; Reactive Oxygen Species; Receptor-Interacting Protein Serine-Threonine Kinases; Ventricular Dysfunction, Left; Ventricular Function, Left

2018
Sodium tanshinone IIA sulfate protects myocardium against paraquat-induced toxicity through activating the Nrf2 signaling pathway in rats.
    Human & experimental toxicology, 2019, Volume: 38, Issue:2

    Topics: Abietanes; Animals; Apoptosis; Cardiotoxicity; Herbicides; Male; Myocardium; NF-E2-Related Factor 2; Paraquat; Protective Agents; Proto-Oncogene Proteins c-bcl-2; Rats, Sprague-Dawley; Signal Transduction

2019
Cardiac-Specific Knockout of ETA Receptor Mitigates Paraquat-Induced Cardiac Contractile Dysfunction.
    Cardiovascular toxicology, 2016, Volume: 16, Issue:3

    Topics: Animals; Apoptosis; Calcium Signaling; Cardiotoxicity; Disease Models, Animal; Female; Heart Diseases; HSP90 Heat-Shock Proteins; Male; Mice, Knockout; Mitochondria, Heart; Myocardial Contraction; Myocytes, Cardiac; Oxidative Phosphorylation; Paraquat; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha; Receptor, Endothelin A; Uncoupling Protein 2; Ventricular Function, Left

2016
Toll-like receptor 4 knockout alleviates paraquat-induced cardiomyocyte contractile dysfunction through an autophagy-dependent mechanism.
    Toxicology letters, 2016, Aug-22, Volume: 257

    Topics: AMP-Activated Protein Kinases; Animals; Autophagy; Autophagy-Related Protein-1 Homolog; Calcium Signaling; Cardiotoxicity; Genotype; Heart Diseases; Herbicides; Mice, Knockout; Myocardial Contraction; Myocytes, Cardiac; Paraquat; Phenotype; Toll-Like Receptor 4; TOR Serine-Threonine Kinases

2016