palmitoyl coenzyme a has been researched along with Innate Inflammatory Response in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Belsham, DD; Tse, EK | 1 |
| Barnhart, S; Bornfeldt, KE; Coleman, RA; Gonzalez, O; Handa, P; Kanter, JE; Kim, F; Kramer, F; Li, X; Pennathur, S; Schaffer, JE; Shen, X; Tsuchiya, K; Vivekanandan-Giri, A | 1 |
| Andrews, G; Colville-Nash, P; Freemantle, C; Lam, C; Papworth, J; Willis, D; Willoughby, D | 1 |
3 other study(ies) available for palmitoyl coenzyme a and Innate Inflammatory Response
| Article | Year |
|---|---|
Palmitate induces neuroinflammation, ER stress, and Pomc mRNA expression in hypothalamic mHypoA-POMC/GFP neurons through novel mechanisms that are prevented by oleate.
Topics: Animals; Biomarkers; Ceramides; Endoplasmic Reticulum Stress; Gene Expression Regulation; Green Fluorescent Proteins; Hypothalamus; I-kappa B Kinase; Inflammation; Insulin; Male; MAP Kinase Signaling System; Mice, Transgenic; Models, Biological; Neurons; Oleic Acid; Palmitates; Palmitoyl Coenzyme A; Pro-Opiomelanocortin; RNA, Messenger; Toll-Like Receptor 4 | 2018 |
Endothelial acyl-CoA synthetase 1 is not required for inflammatory and apoptotic effects of a saturated fatty acid-rich environment.
Topics: Acyl Coenzyme A; Adipose Tissue; Animals; Aorta; Apoptosis; Cattle; Cells, Cultured; Coenzyme A Ligases; Disease Models, Animal; Endoplasmic Reticulum Stress; Endothelial Cells; Enzyme Activation; Fatty Acids; Inflammation; Insulin Resistance; Intercellular Adhesion Molecule-1; JNK Mitogen-Activated Protein Kinases; Macrophages; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Obesity; Palmitoyl Coenzyme A; RNA Interference; Time Factors; Transfection; Vascular Cell Adhesion Molecule-1 | 2013 |
The peroxisome proliferator-activated receptor alpha activator, Wy14,643, is anti-inflammatory in vivo.
Topics: Administration, Oral; Animals; Anti-Inflammatory Agents; Arachidonate 5-Lipoxygenase; Dinoprostone; Disease Models, Animal; Dose-Response Relationship, Drug; Ear, External; Edema; Female; Indoles; Indomethacin; Inflammation; Leukotriene B4; Lipoxygenase Inhibitors; Mice; Neutrophils; Palmitoyl Coenzyme A; Peroxisomes; PPAR alpha; Prostaglandin-Endoperoxide Synthases; Pyrimidines | 2005 |