palmitic acid has been researched along with Hypertrophy in 9 studies
Palmitic Acid: A common saturated fatty acid found in fats and waxes including olive oil, palm oil, and body lipids.
hexadecanoic acid : A straight-chain, sixteen-carbon, saturated long-chain fatty acid.
Hypertrophy: General increase in bulk of a part or organ due to CELL ENLARGEMENT and accumulation of FLUIDS AND SECRETIONS, not due to tumor formation, nor to an increase in the number of cells (HYPERPLASIA).
| Excerpt | Relevance | Reference |
|---|---|---|
| " Cells exposed to angiotensin II remained viable and did not show signs of hypertrophy." | 7.74 | Functional coupling of angiotensin II type 1 receptor with insulin resistance of energy substrate uptakes in immortalized cardiomyocytes (HL-1 cells). ( Alfarano, C; Cerbai, E; Mannucci, E; Mugelli, A; Nediani, C; Raimondi, L; Sartiani, L, 2008) |
| "Cardiac hypertrophy is one of the important pathological features of PA-induced lipotoxicity, but the mechanism by which PA induces cardiomyocyte hypertrophy is still unclear." | 5.43 | Inhibition of Receptor Interacting Protein Kinases Attenuates Cardiomyocyte Hypertrophy Induced by Palmitic Acid. ( Bao, Q; Chai, H; Chen, L; Lei, S; Liu, X; Lu, L; Tang, X; Wu, S; Wu, W; Zhao, M, 2016) |
| " Herein, we evaluated the in vitro protective effects of an ACN rich extract against palmitic acid (PA)-induced hypertrophy, inflammation, and insulin resistance in 3T3-L1 adipocytes." | 3.91 | Anthocyanins ameliorate palmitate-induced inflammation and insulin resistance in 3T3-L1 adipocytes. ( Bashllari, R; Cimino, F; Cristani, M; Molonia, MS; Muscarà, C; Occhiuto, C; Saija, A; Speciale, A, 2019) |
| "Although mutations in the gamma-subunit of AMP-activated protein kinase (AMPK) can result in excessive glycogen accumulation and cardiac hypertrophy, the mechanisms by which this occurs have not been well defined." | 3.74 | The AMPK gamma1 R70Q mutant regulates multiple metabolic and growth pathways in neonatal cardiac myocytes. ( Allard, MF; Dyck, JR; Folmes, KD; Witters, LA; Young, ME, 2007) |
| " Cells exposed to angiotensin II remained viable and did not show signs of hypertrophy." | 3.74 | Functional coupling of angiotensin II type 1 receptor with insulin resistance of energy substrate uptakes in immortalized cardiomyocytes (HL-1 cells). ( Alfarano, C; Cerbai, E; Mannucci, E; Mugelli, A; Nediani, C; Raimondi, L; Sartiani, L, 2008) |
| "Cardiac hypertrophy is one of the important pathological features of PA-induced lipotoxicity, but the mechanism by which PA induces cardiomyocyte hypertrophy is still unclear." | 1.43 | Inhibition of Receptor Interacting Protein Kinases Attenuates Cardiomyocyte Hypertrophy Induced by Palmitic Acid. ( Bao, Q; Chai, H; Chen, L; Lei, S; Liu, X; Lu, L; Tang, X; Wu, S; Wu, W; Zhao, M, 2016) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (44.44) | 29.6817 |
| 2010's | 4 (44.44) | 24.3611 |
| 2020's | 1 (11.11) | 2.80 |
| Authors | Studies |
|---|---|
| Molonia, MS | 2 |
| Occhiuto, C | 2 |
| Muscarà, C | 2 |
| Speciale, A | 2 |
| Ruberto, G | 1 |
| Siracusa, L | 1 |
| Cristani, M | 2 |
| Saija, A | 2 |
| Cimino, F | 2 |
| Hammarstedt, A | 1 |
| Syed, I | 1 |
| Vijayakumar, A | 1 |
| Eliasson, B | 1 |
| Gogg, S | 1 |
| Kahn, BB | 1 |
| Smith, U | 1 |
| Luo, Y | 1 |
| Wu, MY | 1 |
| Deng, BQ | 1 |
| Huang, J | 1 |
| Hwang, SH | 1 |
| Li, MY | 1 |
| Zhou, CY | 1 |
| Zhang, QY | 1 |
| Yu, HB | 1 |
| Zhao, DK | 1 |
| Zhang, G | 1 |
| Qin, L | 1 |
| Peng, A | 1 |
| Hammock, BD | 1 |
| Liu, JY | 1 |
| Bashllari, R | 1 |
| Zhao, M | 1 |
| Lu, L | 1 |
| Lei, S | 1 |
| Chai, H | 1 |
| Wu, S | 1 |
| Tang, X | 1 |
| Bao, Q | 1 |
| Chen, L | 1 |
| Wu, W | 1 |
| Liu, X | 1 |
| Perona, JS | 1 |
| Ruiz-Gutierrez, V | 1 |
| Folmes, KD | 1 |
| Witters, LA | 1 |
| Allard, MF | 1 |
| Young, ME | 1 |
| Dyck, JR | 1 |
| Alfarano, C | 1 |
| Sartiani, L | 1 |
| Nediani, C | 1 |
| Mannucci, E | 1 |
| Mugelli, A | 1 |
| Cerbai, E | 1 |
| Raimondi, L | 1 |
| Wang, Q | 1 |
| Donthi, RV | 1 |
| Wang, J | 1 |
| Lange, AJ | 1 |
| Watson, LJ | 1 |
| Jones, SP | 1 |
| Epstein, PN | 1 |
9 other studies available for palmitic acid and Hypertrophy
| Article | Year |
|---|---|
Effects of a pinitol-rich
Topics: Adipocytes; Animals; Glycyrrhiza; Humans; Hypertrophy; Inflammation; Inositol; Insulin; Insulin Resi | 2022 |
Adipose tissue dysfunction is associated with low levels of the novel Palmitic Acid Hydroxystearic Acids.
Topics: 3T3-L1 Cells; Adipocytes; Adipogenesis; Adult; Animals; Female; Gene Silencing; Glucose Transporter | 2018 |
Inhibition of soluble epoxide hydrolase attenuates a high-fat diet-mediated renal injury by activating PAX2 and AMPK.
Topics: Adenylate Kinase; Animals; Cytochrome P-450 Enzyme System; Diet, High-Fat; Disease Models, Animal; E | 2019 |
Anthocyanins ameliorate palmitate-induced inflammation and insulin resistance in 3T3-L1 adipocytes.
Topics: 3T3-L1 Cells; Adipocytes; Animals; Anthocyanins; Anti-Inflammatory Agents; Hypertrophy; Insulin Resi | 2019 |
Inhibition of Receptor Interacting Protein Kinases Attenuates Cardiomyocyte Hypertrophy Induced by Palmitic Acid.
Topics: Animals; Apoptosis; Blotting, Western; Cell Line; Down-Regulation; Endoplasmic Reticulum Stress; Hyp | 2016 |
Triacylglycerol molecular species are depleted to different extents in the myocardium of spontaneously hypertensive rats fed two oleic acid-rich oils.
Topics: Animals; Cholesterol; Disease Models, Animal; Eicosanoic Acids; Hypertension; Hypertrophy; Lipoprote | 2005 |
The AMPK gamma1 R70Q mutant regulates multiple metabolic and growth pathways in neonatal cardiac myocytes.
Topics: Active Transport, Cell Nucleus; AMP-Activated Protein Kinases; Animals; Animals, Newborn; Cardiomega | 2007 |
Functional coupling of angiotensin II type 1 receptor with insulin resistance of energy substrate uptakes in immortalized cardiomyocytes (HL-1 cells).
Topics: Angiotensin II; Angiotensin II Type 1 Receptor Blockers; Animals; Cell Line; Cell Survival; Glucose; | 2008 |
Cardiac phosphatase-deficient 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase increases glycolysis, hypertrophy, and myocyte resistance to hypoxia.
Topics: Animals; Cell Hypoxia; Disease Models, Animal; Glycolysis; Hypertrophy; Insulin; Male; Mice; Mice, T | 2008 |