palmidrol has been researched along with Brain Ischemia in 8 studies
palmidrol: a cannabinoid receptor-inactive eCB-related molecule used as prophylactic in helping to prevent respiratory viral infection
palmitoyl ethanolamide : An N-(long-chain-acyl)ethanolamine that is the ethanolamide of palmitic (hexadecanoic) acid.
Brain Ischemia: Localized reduction of blood flow to brain tissue due to arterial obstruction or systemic hypoperfusion. This frequently occurs in conjunction with brain hypoxia (HYPOXIA, BRAIN). Prolonged ischemia is associated with BRAIN INFARCTION.
| Excerpt | Relevance | Reference |
|---|---|---|
| "The combination of palmitoylethanolamide (PEA), an endogenous fatty acid amide belonging to the family of the N-acylethanolamines, and the flavonoid luteolin has been found to exert neuroprotective activities in a variety of mouse models of neurological disorders, including brain ischemia." | 7.83 | PEA and luteolin synergistically reduce mast cell-mediated toxicity and elicit neuroprotection in cell-based models of brain ischemia. ( Benarese, M; Fusco, M; Iorio, R; Lanzillotta, A; Mota, M; Parrella, E; Pizzi, M; Porrini, V; Spano, P; Tonin, P, 2016) |
| "The combination of palmitoylethanolamide (PEA), an endogenous fatty acid amide belonging to the family of the N-acylethanolamines, and the flavonoid luteolin has been found to exert neuroprotective activities in a variety of mouse models of neurological disorders, including brain ischemia." | 3.83 | PEA and luteolin synergistically reduce mast cell-mediated toxicity and elicit neuroprotection in cell-based models of brain ischemia. ( Benarese, M; Fusco, M; Iorio, R; Lanzillotta, A; Mota, M; Parrella, E; Pizzi, M; Porrini, V; Spano, P; Tonin, P, 2016) |
| "Focal cerebral ischemia was induced by transient middle cerebral artery occlusion (MCAo) in the right hemisphere." | 1.51 | ( Cordaro, M; Crupi, R; Cuzzocrea, S; D'Amico, R; Di Paola, R; Fusco, R; Gugliandolo, E; Impellizzeri, D; Peritore, AF; Scuto, M; Siracusa, R, 2019) |
| "Focal cerebral ischemia (FCI) induces rapid neuronal death in the ischemic core, which gradually expands toward the penumbra, partly as the result of a neuroinflammatory response." | 1.32 | Palmitoylethanolamide increases after focal cerebral ischemia and potentiates microglial cell motility. ( Franklin, A; Greenberg, DA; Parmentier-Batteur, S; Stella, N; Walter, L, 2003) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (25.00) | 29.6817 |
| 2010's | 5 (62.50) | 24.3611 |
| 2020's | 1 (12.50) | 2.80 |
| Authors | Studies |
|---|---|
| Fusco, R | 1 |
| Scuto, M | 1 |
| Cordaro, M | 3 |
| D'Amico, R | 1 |
| Gugliandolo, E | 1 |
| Siracusa, R | 1 |
| Peritore, AF | 1 |
| Crupi, R | 1 |
| Impellizzeri, D | 1 |
| Cuzzocrea, S | 3 |
| Di Paola, R | 2 |
| Kong, D | 1 |
| Xie, B | 1 |
| Li, Y | 1 |
| Xu, Y | 1 |
| Esposito, E | 2 |
| Caltagirone, C | 1 |
| Cisari, C | 1 |
| Schievano, C | 1 |
| Bruschetta, G | 1 |
| Parrella, E | 1 |
| Porrini, V | 1 |
| Iorio, R | 1 |
| Benarese, M | 1 |
| Lanzillotta, A | 1 |
| Mota, M | 1 |
| Fusco, M | 1 |
| Tonin, P | 1 |
| Spano, P | 1 |
| Pizzi, M | 1 |
| Quartu, M | 1 |
| Poddighe, L | 1 |
| Melis, T | 1 |
| Serra, MP | 1 |
| Boi, M | 1 |
| Lisai, S | 1 |
| Carta, G | 1 |
| Murru, E | 1 |
| Muredda, L | 1 |
| Collu, M | 1 |
| Banni, S | 1 |
| Schäbitz, WR | 1 |
| Giuffrida, A | 1 |
| Berger, C | 1 |
| Aschoff, A | 1 |
| Schwaninger, M | 1 |
| Schwab, S | 1 |
| Piomelli, D | 1 |
| Franklin, A | 1 |
| Parmentier-Batteur, S | 1 |
| Walter, L | 1 |
| Greenberg, DA | 1 |
| Stella, N | 1 |
1 review available for palmidrol and Brain Ischemia
| Article | Year |
|---|---|
Roles of fatty acid ethanolamides (FAE) in traumatic and ischemic brain injury.
Topics: Amides; Animals; Brain; Brain Injuries; Brain Ischemia; Ethanolamines; Humans; Palmitic Acids; Strok | 2014 |
7 other studies available for palmidrol and Brain Ischemia
| Article | Year |
|---|---|
Topics: Amides; Animals; Apoptosis; Biomarkers; Brain Ischemia; Brain-Derived Neurotrophic Factor; Cell Degr | 2019 |
PEA prevented early BBB disruption after cerebral ischaemic/reperfusion (I/R) injury through regulation of ROCK/MLC signaling.
Topics: Amides; Animals; Blood-Brain Barrier; Brain Ischemia; Cell Line; Ethanolamines; Humans; Mice; Myosin | 2021 |
Co-ultramicronized Palmitoylethanolamide/Luteolin in the Treatment of Cerebral Ischemia: from Rodent to Man.
Topics: Adult; Aged; Aged, 80 and over; Amides; Animals; Antioxidants; Behavior, Animal; Brain Ischemia; Coh | 2016 |
PEA and luteolin synergistically reduce mast cell-mediated toxicity and elicit neuroprotection in cell-based models of brain ischemia.
Topics: Amides; Animals; Brain Ischemia; Cell Degranulation; Cells, Cultured; Coculture Techniques; Ethanola | 2016 |
Involvement of the endocannabinoid system in the physiological response to transient common carotid artery occlusion and reperfusion.
Topics: Amides; Animals; Arachidonic Acids; Brain Ischemia; Carotid Artery, Common; Cerebrovascular Disorder | 2017 |
Release of fatty acid amides in a patient with hemispheric stroke: a microdialysis study.
Topics: Aged; Amides; Arachidonic Acids; Brain; Brain Chemistry; Brain Ischemia; Endocannabinoids; Ethanolam | 2002 |
Palmitoylethanolamide increases after focal cerebral ischemia and potentiates microglial cell motility.
Topics: Amides; Animals; Arachidonic Acids; Brain Ischemia; Cannabinoid Receptor Modulators; Cannabinoids; C | 2003 |