Page last updated: 2024-11-02

oxidopamine and Herpes Simplex Virus Infection

oxidopamine has been researched along with Herpes Simplex Virus Infection in 3 studies

Oxidopamine: A neurotransmitter analogue that depletes noradrenergic stores in nerve endings and induces a reduction of dopamine levels in the brain. Its mechanism of action is related to the production of cytolytic free-radicals.
oxidopamine : A benzenetriol that is phenethylamine in which the hydrogens at positions 2, 4, and 5 on the phenyl ring are replaced by hydroxy groups. It occurs naturally in human urine, but is also produced as a metabolite of the drug DOPA (used for the treatment of Parkinson's disease).

Research Excerpts

ExcerptRelevanceReference
" Using the glucocorticoid receptor antagonist RU486, the mineralocorticoid receptor antagonist spironolactone, and the beta-adrenergic receptor antagonist nadolol, the roles of corticosterone and norepinephrine in sympathectomy-mediated modulation of both the primary and memory cellular immune responses to herpes simplex virus type 1 (HSV-1) infection was investigated."3.70Mechanisms underlying chemical sympathectomy-induced suppression of herpes simplex virus-specific cytotoxic T lymphocyte activation and function. ( Bonneau, RH; Leo, NA, 2000)

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19901 (33.33)18.7374
1990's1 (33.33)18.2507
2000's1 (33.33)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Leo, NA1
Bonneau, RH1
Gordon, YJ1
McKnight, JL1
Ostrove, JM1
Romanowski, E1
Araullo-Cruz, T1
Price, RW1

Other Studies

3 other studies available for oxidopamine and Herpes Simplex Virus Infection

ArticleYear
Mechanisms underlying chemical sympathectomy-induced suppression of herpes simplex virus-specific cytotoxic T lymphocyte activation and function.
    Journal of neuroimmunology, 2000, Oct-02, Volume: 110, Issue:1-2

    Topics: Animals; Cell Line; Corticosterone; Herpes Simplex; Herpesvirus 1, Human; Hormone Antagonists; Lymph

2000
Host species and strain differences affect the ability of an HSV-1 ICP0 deletion mutant to establish latency and spontaneously reactivate in vivo.
    Virology, 1990, Volume: 178, Issue:2

    Topics: Animals; Cells, Cultured; Eye Infections, Viral; Herpes Simplex; Host-Parasite Interactions; Hydroxy

1990
Herpes simplex virus latency: adaptation to the peripheral nervous system. I.
    Cancer investigation, 1985, Volume: 3, Issue:3

    Topics: Adaptation, Physiological; Axonal Transport; Axons; Culture Techniques; Epithelium; Herpes Simplex;

1985