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oxidopamine and Delayed Effects, Prenatal Exposure

oxidopamine has been researched along with Delayed Effects, Prenatal Exposure in 15 studies

Oxidopamine: A neurotransmitter analogue that depletes noradrenergic stores in nerve endings and induces a reduction of dopamine levels in the brain. Its mechanism of action is related to the production of cytolytic free-radicals.
oxidopamine : A benzenetriol that is phenethylamine in which the hydrogens at positions 2, 4, and 5 on the phenyl ring are replaced by hydroxy groups. It occurs naturally in human urine, but is also produced as a metabolite of the drug DOPA (used for the treatment of Parkinson's disease).

Research

Studies (15)

TimeframeStudies, this research(%)All Research%
pre-19903 (20.00)18.7374
1990's2 (13.33)18.2507
2000's6 (40.00)29.6817
2010's4 (26.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Mohamed Moosa, Z1
Daniels, WM2
Mabandla, MV2
Baier, CJ1
Pallarés, ME1
Adrover, E1
Katunar, MR1
Raisman-Vozari, R1
Antonelli, MC1
Bałasz, M1
Szkilnik, R2
Brus, R2
Malinowska-Borowska, J1
Kasperczyk, S1
Nowak, D1
Kostrzewa, RM2
Nowak, P2
Kellaway, LA1
Russell, VA1
Bojanek, K1
Jośko, J1
Boroń, D1
Adwent, M1
Gorczyca, P1
Tanyel, FC1
Ertunç, M1
Ekinci, S1
Otçu, S1
Yildirim, M1
Onur, R1
Shabanov, PD2
Lebedev, AA2
Meshcherov, ShK2
Strel'tsov, VF2
Ling, ZD1
Chang, Q1
Lipton, JW1
Tong, CW1
Landers, TM1
Carvey, PM1
Dong, Y1
Chen, Y1
Wang, Z1
Naito, J1
Chen, JL1
Slotkin, TA1
Feigenbaum, JJ1
Yanai, J2
Gorio, A1
Vergani, L1
Malosio, ML1
Lesma, E1
Di Giulio, AM1
González-Ariki, S1
Husband, AJ1
Laxer, U1
Pick, CG1
Trombka, D1

Reviews

2 reviews available for oxidopamine and Delayed Effects, Prenatal Exposure

ArticleYear
The cardiac-sympathetic axis as a teratological model.
    Progress in clinical and biological research, 1983, Volume: 140

    Topics: Animals; Animals, Newborn; Catecholamines; Central Nervous System; Drug-Related Side Effects and Adv

1983
Evidence for the involvement of central dopaminergic receptors in the acute and chronic effects induced by barbiturates.
    Neuropsychobiology, 1983, Volume: 9, Issue:2-3

    Topics: Adenylyl Cyclases; Animals; Barbiturates; Brain; Chlorpromazine; Dopamine; Drug Interactions; Drug T

1983

Other Studies

13 other studies available for oxidopamine and Delayed Effects, Prenatal Exposure

ArticleYear
The effects of prenatal methylmercury exposure on trace element and antioxidant levels in rats following 6-hydroxydopamine-induced neuronal insult.
    Metabolic brain disease, 2014, Volume: 29, Issue:2

    Topics: Animals; Antioxidants; Biomarkers; Brain; Female; Male; Methylmercury Compounds; Neurons; Oxidopamin

2014
Intrastriatal 6-OHDA lesion differentially affects dopaminergic neurons in the ventral tegmental area of prenatally stressed rats.
    Neurotoxicity research, 2014, Volume: 26, Issue:3

    Topics: Animals; Corpus Striatum; Dopaminergic Neurons; Female; Male; Nitric Oxide Synthase Type I; Nucleus

2014
Perinatal manganese exposure and hydroxyl radical formation in rat brain.
    Neurotoxicity research, 2015, Volume: 27, Issue:1

    Topics: Animals; Brain; Brain Chemistry; Female; Frontal Lobe; Hippocampus; Hydroxyl Radical; Male; Manganes

2015
Effect of exercise on dopamine neuron survival in prenatally stressed rats.
    Metabolic brain disease, 2009, Volume: 24, Issue:4

    Topics: Animals; Cell Survival; Denervation; Disease Models, Animal; Dopamine; Exercise Therapy; Female; For

2009
Ontogenetic exposure of rats to pre- and post-natal manganese enhances behavioral impairments produced by perinatal 6-hydroxydopamine.
    Neurotoxicity research, 2011, Volume: 19, Issue:4

    Topics: Animals; Animals, Newborn; Catalepsy; Drug Synergism; Female; Manganese; Oxidopamine; Pregnancy; Pre

2011
Chemical sympathectomy by 6-OH dopamine during fetal life results in inguinal testis through altering cremasteric contractility in rats.
    Journal of pediatric surgery, 2003, Volume: 38, Issue:11

    Topics: Animals; Calcium Channel Blockers; Cryptorchidism; Enzyme Inhibitors; Female; Isoproterenol; Male; M

2003
[The effect of neurochemical damage of dopaminergic terminals in early ontogenesis on the behavior of adult rats].
    Rossiiskii fiziologicheskii zhurnal imeni I.M. Sechenova, 2003, Volume: 89, Issue:11

    Topics: Animals; Animals, Newborn; Behavior, Animal; Brain; Dopamine; Female; Male; Maze Learning; Memory, S

2003
Combined toxicity of prenatal bacterial endotoxin exposure and postnatal 6-hydroxydopamine in the adult rat midbrain.
    Neuroscience, 2004, Volume: 124, Issue:3

    Topics: Animals; Animals, Newborn; Cell Death; Disease Models, Animal; Dopamine; Encephalitis; Endotoxins; F

2004
The effects of neurochemical lesioning of dopaminergic terminals in early ontogenesis on behavior in adult rats.
    Neuroscience and behavioral physiology, 2005, Volume: 35, Issue:5

    Topics: Age Factors; Animals; Animals, Newborn; Behavior, Animal; Brain Injuries; Conditioning, Operant; Dop

2005
Role of sympathetic nerves on early embryonic development and immune modulation of uterus in pregnant mice.
    Autonomic neuroscience : basic & clinical, 2007, Jan-30, Volume: 131, Issue:1-2

    Topics: Age Factors; Animals; CD4-Positive T-Lymphocytes; CD8-Positive T-Lymphocytes; Cell Count; Embryo, Ma

2007
Perinatal exposure to morphine: reactive changes in the brain after 6-hydroxydopamine.
    European journal of pharmacology, 1996, May-06, Volume: 303, Issue:1-2

    Topics: Adrenergic Agents; Animals; Animals, Newborn; Brain; Caudate Nucleus; Female; Mesencephalon; Morphin

1996
Sympathectomy abrogates immunodeficiency associated with protein-energy malnutrition.
    Journal of neuroimmunology, 1999, Sep-01, Volume: 99, Issue:1

    Topics: Animals; Female; Immunoglobulin A; Immunologic Deficiency Syndromes; Injections, Intraperitoneal; In

1999
Dopaminergic denervation reverses behavioral deficits induced by prenatal exposure to phenobarbital.
    Brain research. Developmental brain research, 1989, Aug-01, Volume: 48, Issue:2

    Topics: Animals; Cholinergic Fibers; Dopamine; Female; Hippocampus; Hydroxydopamines; Learning Disabilities;

1989