oxidopamine has been researched along with Brain Damage, Chronic in 6 studies
Oxidopamine: A neurotransmitter analogue that depletes noradrenergic stores in nerve endings and induces a reduction of dopamine levels in the brain. Its mechanism of action is related to the production of cytolytic free-radicals.
oxidopamine : A benzenetriol that is phenethylamine in which the hydrogens at positions 2, 4, and 5 on the phenyl ring are replaced by hydroxy groups. It occurs naturally in human urine, but is also produced as a metabolite of the drug DOPA (used for the treatment of Parkinson's disease).
Brain Damage, Chronic: A condition characterized by long-standing brain dysfunction or damage, usually of three months duration or longer. Potential etiologies include BRAIN INFARCTION; certain NEURODEGENERATIVE DISORDERS; CRANIOCEREBRAL TRAUMA; ANOXIA, BRAIN; ENCEPHALITIS; certain NEUROTOXICITY SYNDROMES; metabolic disorders (see BRAIN DISEASES, METABOLIC); and other conditions.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (16.67) | 18.7374 |
| 1990's | 1 (16.67) | 18.2507 |
| 2000's | 4 (66.67) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Urakawa, S | 1 |
| Hida, H | 1 |
| Masuda, T | 1 |
| Misumi, S | 1 |
| Kim, TS | 1 |
| Nishino, H | 1 |
| Yang, J | 1 |
| Sadler, TR | 1 |
| Givrad, TK | 1 |
| Maarek, JM | 1 |
| Holschneider, DP | 1 |
| Katoh-Semba, R | 1 |
| Tsuzuki, M | 1 |
| Miyazaki, N | 1 |
| Yoshida, A | 1 |
| Nakajima, H | 1 |
| Nakagawa, C | 1 |
| Kitajima, S | 1 |
| Matsuda, M | 1 |
| Da Cunha, C | 1 |
| Wietzikoski, EC | 1 |
| Ferro, MM | 1 |
| Martinez, GR | 1 |
| Vital, MA | 1 |
| Hipólide, D | 1 |
| Tufik, S | 1 |
| Canteras, NS | 1 |
| Kopyov, OV | 1 |
| Jacques, DB | 1 |
| Rand, RW | 1 |
| Craft, J | 1 |
| Buckwalter, JG | 1 |
| Bruno, JP | 1 |
| Zigmond, MJ | 1 |
| Stricker, EM | 1 |
6 other studies available for oxidopamine and Brain Damage, Chronic
| Article | Year |
|---|---|
Environmental enrichment brings a beneficial effect on beam walking and enhances the migration of doublecortin-positive cells following striatal lesions in rats.
Topics: Animals; Brain Damage, Chronic; Cell Differentiation; Cell Movement; Cell Proliferation; Corpus Stri | 2007 |
Changes in brain functional activation during resting and locomotor states after unilateral nigrostriatal damage in rats.
Topics: Amphetamine; Animals; Autoradiography; Brain; Brain Damage, Chronic; Brain Mapping; Central Nervous | 2007 |
Distribution and immunohistochemical localization of GDNF protein in selected neural and non-neural tissues of rats during development and changes in unilateral 6-hydroxydopamine lesions.
Topics: Animals; Animals, Newborn; Astrocytes; Brain; Brain Damage, Chronic; Brain Mapping; Denervation; Gli | 2007 |
Hemiparkinsonian rats rotate toward the side with the weaker dopaminergic neurotransmission.
Topics: Analysis of Variance; Animals; Apomorphine; Brain Damage, Chronic; Disease Models, Animal; Dopamine; | 2008 |
Fetal human and pig mesencephalon xenografts have equal effectiveness in behavioral restoration of damaged rat brain.
Topics: Analysis of Variance; Animals; Brain Damage, Chronic; Brain Tissue Transplantation; Fetal Tissue Tra | 1992 |
Rats given dopamine-depleting brain lesions as neonates do not respond to acute homeostatic imbalances as adults.
Topics: Adaptation, Physiological; Animals; Animals, Newborn; Brain; Brain Damage, Chronic; Dopamine; Drinki | 1986 |