oxidopamine has been researched along with Axon Reaction in 4 studies
Oxidopamine: A neurotransmitter analogue that depletes noradrenergic stores in nerve endings and induces a reduction of dopamine levels in the brain. Its mechanism of action is related to the production of cytolytic free-radicals.
oxidopamine : A benzenetriol that is phenethylamine in which the hydrogens at positions 2, 4, and 5 on the phenyl ring are replaced by hydroxy groups. It occurs naturally in human urine, but is also produced as a metabolite of the drug DOPA (used for the treatment of Parkinson's disease).
| Excerpt | Relevance | Reference |
|---|---|---|
| "Huntington's disease is an autosomal dominant hereditary neurodegenerative disorder characterized by severe striatal cell loss." | 1.31 | Mice transgenic for exon 1 of the Huntington's disease gene display reduced striatal sensitivity to neurotoxicity induced by dopamine and 6-hydroxydopamine. ( Brundin, P; Castilho, RF; DiFiglia, M; Hansson, O; Petersén, A; Przedborski, S; Puschban, Z; Rice, M; Romero, N; Sapp, E; Sulzer, D, 2001) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (25.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Ries, V | 1 |
| Silva, RM | 1 |
| Oo, TF | 2 |
| Cheng, HC | 2 |
| Rzhetskaya, M | 2 |
| Kholodilov, N | 2 |
| Flavell, RA | 1 |
| Kuan, CY | 1 |
| Rakic, P | 1 |
| Burke, RE | 2 |
| Kim, SR | 1 |
| Kareva, T | 1 |
| Yarygina, O | 1 |
| Wang, C | 1 |
| During, M | 1 |
| Talloczy, Z | 1 |
| Tanaka, K | 1 |
| Komatsu, M | 1 |
| Kobayashi, K | 1 |
| Okano, H | 1 |
| Sauer, H | 1 |
| Rosenblad, C | 1 |
| Björklund, A | 1 |
| Petersén, A | 1 |
| Hansson, O | 1 |
| Puschban, Z | 1 |
| Sapp, E | 1 |
| Romero, N | 1 |
| Castilho, RF | 1 |
| Sulzer, D | 1 |
| Rice, M | 1 |
| DiFiglia, M | 1 |
| Przedborski, S | 1 |
| Brundin, P | 1 |
4 other studies available for oxidopamine and Axon Reaction
| Article | Year |
|---|---|
JNK2 and JNK3 combined are essential for apoptosis in dopamine neurons of the substantia nigra, but are not required for axon degeneration.
Topics: Animals; Apoptosis; Axons; Dopamine; Gene Expression Regulation; Mice; Mice, Inbred C57BL; Mice, Kno | 2008 |
Akt suppresses retrograde degeneration of dopaminergic axons by inhibition of macroautophagy.
Topics: Animals; Autophagy; Autophagy-Related Protein 7; Axons; Dependovirus; Disease Models, Animal; Dopami | 2011 |
Glial cell line-derived neurotrophic factor but not transforming growth factor beta 3 prevents delayed degeneration of nigral dopaminergic neurons following striatal 6-hydroxydopamine lesion.
Topics: Adrenergic Fibers; Animals; Corpus Striatum; Dopamine; Female; Glial Cell Line-Derived Neurotrophic | 1995 |
Mice transgenic for exon 1 of the Huntington's disease gene display reduced striatal sensitivity to neurotoxicity induced by dopamine and 6-hydroxydopamine.
Topics: Aging; Animals; Ascorbic Acid; Dopamine; Dopamine and cAMP-Regulated Phosphoprotein 32; Dose-Respons | 2001 |