oxalates and Porphyrias

oxalates has been researched along with Porphyrias* in 3 studies

Reviews

1 review(s) available for oxalates and Porphyrias

ArticleYear
[Inborn errors of metabolism and their significance in urology and nephrology].
    Zeitschrift fur Urologie und Nephrologie, 1976, Volume: 69, Issue:5

    Topics: Acidosis, Renal Tubular; Alkaptonuria; Cystinuria; Diabetes Insipidus; Female; Glycine; Gout; Humans; Hyperlipidemias; Hyperparathyroidism; Kidney Diseases; Lesch-Nyhan Syndrome; Male; Metabolism, Inborn Errors; Nephritis, Hereditary; Oxalates; Porphyrias; Uric Acid; Urologic Diseases; Xanthines

1976

Other Studies

2 other study(ies) available for oxalates and Porphyrias

ArticleYear
[Adverse effect of muscle relaxants and narcotics in hereditary diseases (literature survey)].
    Khirurgiia, 1976, Issue:11

    Topics: Arrhythmias, Cardiac; Barbiturates; Butyrylcholinesterase; Drug Synergism; Drug Tolerance; Dysautonomia, Familial; Genetic Diseases, Inborn; Humans; Hyperbilirubinemia, Hereditary; Jaundice; Kidney Calculi; Malignant Hyperthermia; Metabolism, Inborn Errors; Narcotics; Nephrocalcinosis; Osteogenesis Imperfecta; Oxalates; Paralyses, Familial Periodic; Pharmacogenetics; Porphyrias; Succinylcholine

1976
Ascorbate status of patients with porphyria cutanea tarda symptomatica and its effect on porphyrin metabolism.
    The South African journal of medical sciences, 1975, Volume: 40, Issue:4

    The ascorbate status and the effect of loading doses of ascorbic acid (1,5 g per day by mouth for 7 days) on the porphyric process were studied in 7 black men with porphyria cutanea tarda symptomatica. It was found that the ascorbate stores were depleted in these patients as judged by serum and leucocyte ascorbate levels. Temporary repletion of the ascorbate stores was effected by the loading doses of ascorbic acid: Serum iron concentrations increased in 6 patients; urinary iron excretion showed small peaks a few days after ascorbic acid therapy commenced; haemoglobin concentration was not affected; excessive amounts of oxalate were excreted in the urine; neither total urinary porphyrin excretion nor the composition of the urinary porphyrins was affected in a way which could be related to ascorbate therapy. Further evidence that ascorbate depletion is not important in the induction of porphyria was found when the ascorbate status of siderotic rats, rendered porphyric by hexachlorobenzene-feeding, was examined. Stores of ascorbate and the oxidative catabolism of ascorbate were not different in the prophyric rats as compared with normal litter mates.

    Topics: Adult; Animals; Ascorbic Acid; Female; Hemoglobins; Humans; Iron; Leukocytes; Male; Middle Aged; Oxalates; Porphyrias; Porphyrins

1975