oxalates has been researched along with Peripheral-Nervous-System-Diseases* in 6 studies
1 trial(s) available for oxalates and Peripheral-Nervous-System-Diseases
| Article | Year |
|---|---|
Isoniazid-induced vitamin B6 deficiency. Metabolic studies and preliminary vitamin B6 excretion studies.
Topics: Aspartate Aminotransferases; Blood Urea Nitrogen; Erythrocytes; Humans; Isoniazid; Leukocyte Count; Lymphocytes; Niacinamide; Oxalates; Peripheral Nervous System Diseases; Pyridoxine; Tuberculosis, Pulmonary; Vitamin B 6 Deficiency | 1967 |
5 other study(ies) available for oxalates and Peripheral-Nervous-System-Diseases
| Article | Year |
|---|---|
Neurotoxic effect of oxaliplatin: Comparison with its oxalate-free analogue cis-[PtII(1R,2R-DACH)(3-acetoxy-1,1-cyclobutanedicarboxylato)] (LLC-1402) in mice.
Studies suggest that oxalate is involved in the development oxaliplatin-induced peripheral sensory neuropathy (OPSN). This study aimed to compare the neurotoxic effects of oxaliplatin with its oxalate-free cytotoxic analogue cis-[Pt Topics: Animals; Antineoplastic Agents; Male; Mice; Neurotoxicity Syndromes; Organoplatinum Compounds; Oxalates; Oxaliplatin; Peripheral Nervous System Diseases | 2018 |
[Roles of TRPA1 in acute peripheral neuropathy characteristically induced by oxaliplatin].
Topics: Acute Disease; Animals; Antineoplastic Agents; Calcium Channels; Chronic Disease; Humans; Mice; Nerve Tissue Proteins; Organoplatinum Compounds; Oxalates; Oxaliplatin; Peripheral Nervous System Diseases; Transient Receptor Potential Channels; TRPA1 Cation Channel | 2013 |
Peripheral neuropathy in oxalosis. A case report with electron microscopic observations.
A 61 year old man had chronic renal failure because of oxaluria and renal calculi. Two years before death, while on hemodialysis, he developed severe progressive peripheral neuropathy. At autopsy calcium oxalate crystals were found in the peripheral nerves and other tissues. Nerve lesions included segmental demyelination, axonal degeneration and crystalline deposits within the myelin sheath. Ultrastructurally there were foci of osmiophilic granular material within myelin lamellae and endoneurium, and pleomorphic lamellar bodies in the perinuclear Schwann cell cytoplasm. It is probable that chronic hemodialysis favors the deposition of oxalate in the Schwann cells and the development of neuropathy in patients with primary hyperoxaluria and renal failure. Topics: Demyelinating Diseases; Humans; Kidney Calculi; Kidney Failure, Chronic; Male; Median Nerve; Metabolic Diseases; Middle Aged; Nephrectomy; Nerve Fibers, Myelinated; Oxalates; Peripheral Nerves; Peripheral Nervous System Diseases; Radial Nerve; Renal Dialysis; Schwann Cells; Sciatic Nerve | 1976 |
Peripheral neuropathy complicating primary hyperoxaluria.
A patient with chronic renal disease due to primary hyperoxaluria developed a rapidly progressing motor neuropathy with marked impairment of nerve conduction. Pathological studies demonstrated the presence of both axonal degeneration and segmental demyelination, together with the presence of oxalate crystals within axons. It is suggested that the development of peripheral neuropathy complicating hyperoxaluria is a consequence of the increased life-span mad possible by haemodialysis. Topics: Adult; Carbohydrate Metabolism, Inborn Errors; Humans; Kidney Failure, Chronic; Male; Oxalates; Peripheral Nerves; Peripheral Nervous System Diseases; Syndrome | 1976 |
Progressive peripheral neuropathy in patient with primary hyperoxaluria.
In a patient suffering from primary hyperoxaluria with oxalosis a progressive peripheral neuropathy was associated with intra-axonal deposition of microcrystals of calcium oxalate. Probably his neuropathy was the result of mechanical obstruction of axoplasmic flow. Topics: Adult; Autopsy; Axonal Transport; Heart Arrest; Humans; Male; Metabolism, Inborn Errors; Muscles; Oxalates; Paresthesia; Peripheral Nerves; Peripheral Nervous System Diseases; Renal Dialysis; Uremia | 1975 |