ouabain has been researched along with Glioblastoma in 7 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (14.29) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (28.57) | 29.6817 |
| 2010's | 4 (57.14) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Li, D; Liang, F; Yan, X; Zheng, J | 1 |
| Chen, D; Mohamad, O; Song, M; Yu, SP | 1 |
| Chang, HS; Chang, WL; Chen, IS; Guh, JH; Hsu, JL; Hsu, LC; Kung, FL; Leu, WJ; Liu, FL; Yu, CC | 1 |
| El-Mallakh, RS; Huang, X; Lei, Z; Li, XP | 1 |
| Joshi, AD; Parsons, DW; Riggins, GJ; Velculescu, VE | 1 |
| Kurup, PA; Kurup, RK | 1 |
| Palladini, G; Solheid, C | 1 |
7 other study(ies) available for ouabain and Glioblastoma
| Article | Year |
|---|---|
Ouabain elicits human glioblastoma cells apoptosis by generating reactive oxygen species in ERK-p66SHC-dependent pathway.
Topics: Apoptosis; Blotting, Western; Calcium; Caspase 3; Cell Line, Tumor; Cell Survival; Dose-Response Relationship, Drug; Extracellular Signal-Regulated MAP Kinases; Glioblastoma; Humans; Ouabain; Phosphorylation; ras Proteins; Reactive Oxygen Species; RNA Interference; Shc Signaling Adaptor Proteins; Signal Transduction; Src Homology 2 Domain-Containing, Transforming Protein 1; src-Family Kinases; Time Factors | 2015 |
Inhibition of Na+/K+-ATPase induces hybrid cell death and enhanced sensitivity to chemotherapy in human glioblastoma cells.
Topics: Antineoplastic Agents; Apoptosis; Brain Neoplasms; Calcium; Cell Line, Tumor; Dacarbazine; Drug Resistance, Neoplasm; Drug Screening Assays, Antitumor; Gene Knockdown Techniques; Glioblastoma; Humans; Membrane Potential, Mitochondrial; Ouabain; Sodium; Sodium-Potassium-Exchanging ATPase; Temozolomide; Valinomycin | 2014 |
Epi-reevesioside F inhibits Na+/K+-ATPase, causing cytosolic acidification, Bak activation and apoptosis in glioblastoma.
Topics: Antineoplastic Agents; Apoptosis; bcl-2 Homologous Antagonist-Killer Protein; Brain Neoplasms; Calcium; Cell Line, Tumor; Cell Proliferation; Cytosol; Flow Cytometry; Glioblastoma; Humans; Hydrogen-Ion Concentration; Inhibitory Concentration 50; Membrane Potential, Mitochondrial; Ouabain; Potassium; Protein Structure, Tertiary; Rhodamines; Saponins; Sodium; Sodium-Potassium-Exchanging ATPase | 2015 |
Response of sodium pump to ouabain challenge in human glioblastoma cells in culture.
Topics: Antipsychotic Agents; Bipolar Disorder; Blotting, Western; Brain Neoplasms; Cell Line, Tumor; DNA Primers; DNA, Complementary; Enzyme Inhibitors; Glioblastoma; Humans; Ion Transport; Lithium Carbonate; Neuroglia; Nucleic Acid Amplification Techniques; Ouabain; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Sodium-Potassium-Exchanging ATPase; Subcellular Fractions | 2009 |
Sodium ion channel mutations in glioblastoma patients correlate with shorter survival.
Topics: Apoptosis; Brain Neoplasms; Calcium Channels; Cell Line, Tumor; Cell Proliferation; Digoxin; Glioblastoma; Humans; Male; Middle Aged; Mutation; Ouabain; Potassium Channels; Sodium Channels; Survival Analysis | 2011 |
Cerebral chemical dominance and neural regulation of cell division, cell proliferation, neoplastic transformation, and genomic function.
Topics: Adult; Brain; Brain Neoplasms; Cell Division; Cell Membrane; Cell Movement; Cell Transformation, Neoplastic; Digoxin; Down Syndrome; Functional Laterality; Genome, Human; Glioblastoma; Humans; Huntington Disease; Hydroxymethylglutaryl CoA Reductases; Lymphoma, Non-Hodgkin; Magnesium; Neurons; Ouabain | 2003 |
Morphological changes induced by ouabain in normal and neoplastic human glia in monolayer culture.
Topics: Adenosine Triphosphatases; Astrocytoma; Cells, Cultured; Enzyme Activation; Ependymoma; Glioblastoma; Humans; Neuroglia; Ouabain; Potassium; Sodium | 1974 |