ouabain has been researched along with Genetic Predisposition in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (25.00) | 29.6817 |
2010's | 3 (75.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Manunta, P; Pozzoli, S; Simonini, M | 1 |
Koenderink, JB; Messchaert, M; Russel, FG; Swarts, HG; Weigand, KM | 1 |
Koenderink, JB; Russel, FG; Swarts, HG; Weigand, KM | 1 |
Bianchi, G; Burnier, M; Casamassima, N; Citterio, L; Hamlyn, JM; Lanzani, C; Maillard, M; Manunta, P; Simonini, M; Stella, P; Tantardini, C | 1 |
1 review(s) available for ouabain and Genetic Predisposition
Article | Year |
---|---|
Predicting acute kidney injury: current status and future challenges.
Topics: Acute Kidney Injury; Biomarkers; Cystatin C; Early Diagnosis; Fatty Acid-Binding Proteins; Genetic Predisposition to Disease; Genomics; Hepatitis A Virus Cellular Receptor 1; Humans; Insulin-Like Growth Factor Binding Proteins; Interleukin-18; Kidney Function Tests; Lipocalin-2; Models, Biological; Ouabain; Renal Insufficiency, Chronic; Risk Factors; Severity of Illness Index; Tissue Inhibitor of Metalloproteinase-2 | 2018 |
3 other study(ies) available for ouabain and Genetic Predisposition
Article | Year |
---|---|
Alternating Hemiplegia of Childhood mutations have a differential effect on Na(+),K(+)-ATPase activity and ouabain binding.
Topics: Animals; Cell Line; Genetic Predisposition to Disease; Hemiplegia; Humans; Models, Molecular; Mutation; Ouabain; Phenotype; Phosphorylation; Potassium; Protein Binding; Protein Structure, Secondary; Sf9 Cells; Sodium-Potassium-Exchanging ATPase; Spodoptera | 2014 |
Biochemical characterization of sporadic/familial hemiplegic migraine mutations.
Topics: Animals; Cell Line; Cell Membrane; Genetic Predisposition to Disease; Hydrogen Bonding; Migraine with Aura; Models, Molecular; Mutation; Ouabain; Sf9 Cells; Sodium-Potassium-Exchanging ATPase; Spodoptera | 2014 |
Relationships among endogenous ouabain, alpha-adducin polymorphisms and renal sodium handling in primary hypertension.
Topics: Blood Pressure; Blood Pressure Monitoring, Ambulatory; Calmodulin-Binding Proteins; Female; Genetic Predisposition to Disease; Humans; Hypertension; Kidney Tubules; Male; Middle Aged; Ouabain; Polymorphism, Single Nucleotide; Sodium | 2008 |