orabase has been researched along with Necrosis* in 2 studies
2 other study(ies) available for orabase and Necrosis
| Article | Year |
|---|---|
Porous polymethylmethacrylate as bone substitute in the craniofacial area.
In craniofacial surgery, alloplastic materials are used for correcting bony defects. Porous polymethylmethacrylate (PMMA) is a biocompatible and nondegradable bone cement. Porous PMMA is formed by the classic bone cement formulation of methylmethacrylate liquid and PMMA powder in which an aqueous biodegradable carboxymethylcellulose gel is dispersed to create pores in the cement when cured. Pores give bone the opportunity to grow in, resulting in a better fixation of the prostheses. We evaluated the long-term results (n = 14), up to 20 years, of augmentations and defect fillings in the craniofacial area, with special interest in possible side effects and bone ingrowth. The evaluation consisted of a questionnaire, a physical examination, and a computed tomography (CT) scan. There were no side effects that could be ascribed to the porous PMMA. Twelve CT scans showed bone ingrowth into the prostheses, proving the validity behind the concept of porous PMMA. Topics: Biocompatible Materials; Bone Cements; Bone Substitutes; Carboxymethylcellulose Sodium; Facial Bones; Follow-Up Studies; Gels; Humans; Hypersensitivity; Necrosis; Patient Satisfaction; Physical Examination; Polymethyl Methacrylate; Porosity; Prosthesis-Related Infections; Reproducibility of Results; Retrospective Studies; Skull; Staphylococcal Infections; Surface Properties; Surveys and Questionnaires; Tomography, X-Ray Computed; Wound Healing | 2003 |
The protective effect of polyriboinosinic acid-polyribocytidylic acid against the occurrence of galactosamine-induced liver cell injury in rat.
A marked increase of serum transaminase activities, histological changes of livers similar to those seen in viral hepatitis in man, and inhibition of hepatic protein synthesis were observed in rats following a single injection of D-galactosamine-HCl. These galactosamine-induced phenomena were prevented by the pretreatment of polyriboinosinic acid-polyribocytidylic acid 24 h before the galactosamine administration. Topics: Alanine Transaminase; Animals; Aspartate Aminotransferases; Carboxymethylcellulose Sodium; Chemical and Drug Induced Liver Injury; Galactosamine; Liver; Liver Diseases; Male; Methylcellulose; Necrosis; Peptides; Poly I-C; Polylysine; Rats; Rats, Inbred Strains | 1983 |