Page last updated: 2024-11-03

ono 1078 and Necrosis

ono 1078 has been researched along with Necrosis in 2 studies

pranlukast: SRS-A antagonist; leukotriene D4 receptor antagonist

Necrosis: The death of cells in an organ or tissue due to disease, injury or failure of the blood supply.

Research Excerpts

ExcerptRelevanceReference
"Necrosis was the main death mode, and the necrotic rate increased 32% after exposure to 220 μM H2O2 for 4 hours."1.37Pranlukast attenuates hydrogen peroxide-induced necrosis in endothelial cells by inhibiting oxygen reactive species-mediated collapse of mitochondrial membrane potential. ( Fang, SH; Huang, XQ; Lin, KN; Lu, YB; Wei, EQ; Zhang, WP; Zhao, R, 2011)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (50.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Fang, SH2
Yuan, YM1
Peng, F1
Li, CT1
Zhang, LH1
Lu, YB2
Zhang, WP2
Wei, EQ2
Zhao, R1
Lin, KN1
Huang, XQ1

Other Studies

2 other studies available for ono 1078 and Necrosis

ArticleYear
Pranlukast attenuates ischemia-like injury in endothelial cells via inhibiting reactive oxygen species production and nuclear factor-kappaB activation.
    Journal of cardiovascular pharmacology, 2009, Volume: 53, Issue:1

    Topics: Arachidonate 5-Lipoxygenase; Cell Line; Cell Nucleus; Cell Survival; Chromones; Endothelial Cells; G

2009
Pranlukast attenuates hydrogen peroxide-induced necrosis in endothelial cells by inhibiting oxygen reactive species-mediated collapse of mitochondrial membrane potential.
    Journal of cardiovascular pharmacology, 2011, Volume: 57, Issue:4

    Topics: Cell Line; Cell Survival; Chromones; Dose-Response Relationship, Drug; Endothelial Cells; Humans; Hy

2011