Page last updated: 2024-11-03

ono 1078 and Dermatitis, Atopic

ono 1078 has been researched along with Dermatitis, Atopic in 2 studies

pranlukast: SRS-A antagonist; leukotriene D4 receptor antagonist

Dermatitis, Atopic: A chronic inflammatory genetically determined disease of the skin marked by increased ability to form reagin (IgE), with increased susceptibility to allergic rhinitis and asthma, and hereditary disposition to a lowered threshold for pruritus. It is manifested by lichenification, excoriation, and crusting, mainly on the flexural surfaces of the elbow and knee. In infants it is known as infantile eczema.

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Satoh, T1
Tahara, E1
Yamada, T1
Watanabe, C1
Itoh, T1
Terasawa, K1
Nagai, H1
Saiki, I1
Terui, T1
Sano, K1
Okada, M1
Shirota, H1
Honda, M1
Ozawa, M1
Hirasawa, N1
Tamura, G1
Tagami, H1

Other Studies

2 other studies available for ono 1078 and Dermatitis, Atopic

ArticleYear
Differential effect of antiallergic drugs on IgE-mediated cutaneous reaction in passively sensitized mice.
    Pharmacology, 2000, Volume: 60, Issue:2

    Topics: Animals; Anti-Allergic Agents; Antibodies, Monoclonal; Chromones; Dermatitis, Atopic; Dinitrofluorob

2000
Production and pharmacologic modulation of the granulocyte-associated allergic responses to ovalbumin in murine skin models induced by injecting ovalbumin-specific Th1 or Th2 cells.
    The Journal of investigative dermatology, 2001, Volume: 117, Issue:2

    Topics: Animals; Cells, Cultured; Chromones; Dermatitis, Atopic; Disease Models, Animal; Ear; Edema; Enzyme

2001