omeprazole has been researched along with Hormone-Dependent Neoplasms in 1 studies
Omeprazole: A 4-methoxy-3,5-dimethylpyridyl, 5-methoxybenzimidazole derivative of timoprazole that is used in the therapy of STOMACH ULCERS and ZOLLINGER-ELLISON SYNDROME. The drug inhibits an H(+)-K(+)-EXCHANGING ATPASE which is found in GASTRIC PARIETAL CELLS.
omeprazole : A racemate comprising equimolar amounts of (R)- and (S)-omeprazole.
5-methoxy-2-{[(4-methoxy-3,5-dimethylpyridin-2-yl)methyl]sulfinyl}-1H-benzimidazole : A member of the class of benzimidazoles that is 1H-benzimidazole which is substituted by a [4-methoxy-3,5-dimethylpyridin-2-yl)methyl]sulfinyl group at position 2 and a methoxy group at position 5.
| Excerpt | Relevance | Reference |
|---|---|---|
| " Our results indicate that PPIs display tumorigenic activities in PCa cells, suggesting that their long-term administration in patients should be carefully monitored." | 1.51 | Proton pump inhibitors promote the growth of androgen-sensitive prostate cancer cells through ErbB2, ERK1/2, PI3K/Akt, GSK-3β signaling and inhibition of cellular prostatic acid phosphatase. ( Banfi, D; Di Blasio, L; Fanciulli, A; Favaro, E; Gamba, G; Gesmundo, I; Ghigo, E; Gontero, P; Granata, R; Munegato, S; Musuraca, C; Papotti, M; Primo, L; Rapa, I; Villanova, T; Volante, M, 2019) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Gesmundo, I | 1 |
| Di Blasio, L | 1 |
| Banfi, D | 1 |
| Villanova, T | 1 |
| Fanciulli, A | 1 |
| Favaro, E | 1 |
| Gamba, G | 1 |
| Musuraca, C | 1 |
| Rapa, I | 1 |
| Volante, M | 1 |
| Munegato, S | 1 |
| Papotti, M | 1 |
| Gontero, P | 1 |
| Primo, L | 1 |
| Ghigo, E | 1 |
| Granata, R | 1 |
1 other study available for omeprazole and Hormone-Dependent Neoplasms
| Article | Year |
|---|---|
Proton pump inhibitors promote the growth of androgen-sensitive prostate cancer cells through ErbB2, ERK1/2, PI3K/Akt, GSK-3β signaling and inhibition of cellular prostatic acid phosphatase.
Topics: Acid Phosphatase; Animals; Apoptosis; Cell Proliferation; Glycogen Synthase Kinase 3 beta; Humans; M | 2019 |