olmesartan has been researched along with Ventricular Dysfunction, Left in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Haller, H; Menne, J; Ott, C; Raff, U; Ruilope, LM; Schmieder, RE | 1 |
| Ge, J; Guo, J; Jiang, G; Li, L; Wang, S; Wu, J; You, J; Zou, Y | 1 |
| Aoyama, T; Esaki, M; Fujiwara, H; Kanamori, H; Kawasaki, M; Li, Y; Maruyama, R; Minatoguchi, S; Miyata, S; Nakagawa, M; Ogino, A; Okada, H; Takemura, G; Ushikoshi, H | 1 |
1 trial(s) available for olmesartan and Ventricular Dysfunction, Left
| Article | Year |
|---|---|
Prevention of electrocardiographic left ventricular remodeling by the angiotensin receptor blocker olmesartan in patients with type 2 diabetes.
Topics: Adolescent; Adult; Aged; Angiotensin II Type 1 Receptor Blockers; Blood Pressure; Diabetes Mellitus, Type 2; Electrocardiography; Female; Humans; Imidazoles; Male; Middle Aged; Placebos; Tetrazoles; Ventricular Dysfunction, Left; Ventricular Remodeling; Young Adult | 2014 |
2 other study(ies) available for olmesartan and Ventricular Dysfunction, Left
| Article | Year |
|---|---|
Olmesartan attenuates cardiac remodeling through DLL4/Notch1 pathway activation in pressure overload mice.
Topics: Adaptor Proteins, Signal Transducing; Angiotensin II; Angiotensin II Type 1 Receptor Blockers; Animals; Calcium-Binding Proteins; Dipeptides; Disease Models, Animal; Fibrosis; Hypertrophy, Left Ventricular; Imidazoles; Intracellular Signaling Peptides and Proteins; Male; Membrane Proteins; Mice; Mice, Inbred C57BL; Microcirculation; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Receptor, Notch1; Tetrazoles; Up-Regulation; Ventricular Dysfunction, Left; Ventricular Remodeling | 2013 |
Inhibition of Fas-associated apoptosis in granulation tissue cells accompanies attenuation of postinfarction left ventricular remodeling by olmesartan.
Topics: Angiotensin II Type 1 Receptor Blockers; Animals; Apoptosis; fas Receptor; Imidazoles; Male; Mice; Mice, Inbred C57BL; Myocardial Infarction; Tetrazoles; Ventricular Dysfunction, Left; Ventricular Remodeling | 2007 |