oleanolic acid has been researched along with Status Epilepticus in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (75.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Choi, SH; Kang, TC; Kim, JE; Kim, MJ; Kong, MJ; Park, H | 1 |
Kang, TC; Kim, JE; Lee, JE; Park, H | 1 |
Choi, SH; Kang, TC; Kim, JE; Kong, MJ; Park, H | 1 |
Bao, Y; Hu, SJ; Li, HM; Xie, W; Xing, JL; Yu, YH | 1 |
4 other study(ies) available for oleanolic acid and Status Epilepticus
Article | Year |
---|---|
CDDO-Me Attenuates Vasogenic Edema and Astroglial Death by Regulating NF-κB p65 Phosphorylations and Nrf2 Expression Following Status Epilepticus.
Topics: Astrocytes; Brain Edema; Cell Line, Tumor; Gene Expression; Humans; Microglia; NF-E2-Related Factor 2; Nitric Oxide Synthase Type III; Oleanolic Acid; Phosphatidylinositol 3-Kinase; Phosphorylation; Proto-Oncogene Proteins c-akt; Status Epilepticus; Transcription Factor RelA; Tumor Necrosis Factor-alpha | 2019 |
CDDO-Me Inhibits Microglial Activation and Monocyte Infiltration by Abrogating NFκB- and p38 MAPK-Mediated Signaling Pathways Following Status Epilepticus.
Topics: Animals; Chemokine CCL2; Frontal Lobe; Male; MAP Kinase Signaling System; Microglia; Models, Biological; Monocytes; NF-E2-Related Factor 2; NF-kappa B; Oleanolic Acid; p38 Mitogen-Activated Protein Kinases; Parietal Lobe; Peptides; Phosphorylation; Phosphoserine; Rats, Sprague-Dawley; Status Epilepticus; Tumor Necrosis Factor-alpha | 2020 |
CDDO-Me Selectively Attenuates CA1 Neuronal Death Induced by Status Epilepticus via Facilitating Mitochondrial Fission Independent of LONP1.
Topics: Animals; ATP-Dependent Proteases; CA1 Region, Hippocampal; Cell Death; Male; Mitochondrial Dynamics; Neurons; Neuroprotective Agents; Oleanolic Acid; Rats; Rats, Sprague-Dawley; Status Epilepticus | 2019 |
Saikosaponin a mediates the anticonvulsant properties in the HNC models of AE and SE by inhibiting NMDA receptor current and persistent sodium current.
Topics: Animals; Anticonvulsants; Cells, Cultured; Electrophysiology; Epilepsy; Hippocampus; Neurons; Oleanolic Acid; Rats; Rats, Sprague-Dawley; Receptors, N-Methyl-D-Aspartate; Saponins; Sodium; Status Epilepticus | 2012 |